Results 131 to 140 of about 420,555 (284)

Wolbachia infections in native and introduced populations of fire ants (Solenopsis spp.) [PDF]

open access: yes, 2000
Wolbachia are cytoplasmically inherited bacteria that induce a variety of effects with fitness consequences on host arthropods, including cytoplasmic incompatibility, parthenogenesis, male-killing and feminization.
Keller, L.   +4 more
core   +1 more source

PD‐1 Inhibits CD4+ TRM‐Mediated cDC1 Mobilization via Suppressing JAML in Human NSCLC

open access: yesAdvanced Science, EarlyView.
CD4+ tissue‐resident memory T cells (TRMs) in non‐small cell lung cancer recruit conventional type 1 dendritic cells via XCL1‐XCR1 signaling, orchestrating antitumor immunity. The costimulatory molecule JAML is essential for this process. PD‐1 blockade restores JAML expression and cDC1 mobilization, while JAML agonists synergize with anti‐PD‐1 therapy,
Zheyu Shao   +16 more
wiley   +1 more source

Wolbachia divergence and the evolution of cytoplasmic incompatibility in Culex pipiens.

open access: yesPLoS ONE, 2014
Many insect species harbor Wolbachia bacteria that induce cytoplasmic incompatibility (CI), i.e. embryonic lethality in crosses between infected males and uninfected females, or between males and females carrying incompatible Wolbachia strains.
Célestine M Atyame   +6 more
doaj   +1 more source

On maximal chain subgraphs and covers of bipartite graphs [PDF]

open access: yes, 2016
In this paper, we address three related problems. One is the enumeration of all the maximal edge induced chain subgraphs of a bipartite graph, for which we provide a polynomial delay algorithm.
Calamoneri, Tiziana   +4 more
core   +1 more source

Evolution-guided mutagenesis of the cytoplasmic incompatibility proteins: Identifying CifA’s complex functional repertoire and new essential regions in CifB

open access: yesbioRxiv, 2020
Wolbachia are the world’s most common, maternally-inherited, arthropod endosymbionts. Their worldwide distribution is due in part to a selfish drive system termed cytoplasmic incompatibility (CI) that confers a relative fitness advantage to females that ...
J. Shropshire   +2 more
semanticscholar   +1 more source

Ribosome Homeostasis Regulated by SETD2 Preserves Intestinal Epithelial Barrier

open access: yesAdvanced Science, EarlyView.
SETD2 ablation causes dysregulation and recruitment defects of ribosome biogenesis factors, resulting in translational disorders of barrier maintenance genes, thereby compromising the intestinal barrier. These findings unveil a previously unappreciated role of ribosome biogenesis and translational regulation in preserving the intestinal epithelial ...
Hanyu Rao   +11 more
wiley   +1 more source

Structured and unstructured continuous models for Wolbachia infections [PDF]

open access: yes, 2009
We introduce and investigate a series of models for an infection of a diplodiploid host species by the bacterial endosymbiont Wolbachia. The continuous models are characterized by partial vertical transmission, cytoplasmic incompatibility and fitness ...
Farkas, József Z.   +2 more
core  

Melanic mutation causes a fitness decline in bean beetles infected by Wolbachia [PDF]

open access: yes, 2017
Wolbachia cannot live outside a host, which is thought to be the reason for host‐Wolbachia coevolution toward benign parasitism, especially because the fitness of Wolbachia is traded against its host\u27s fitness.
Kondo Natsuko I.   +3 more
core   +2 more sources

Wolbachia and cytoplasmic imcompatibility in mosquitoes

open access: yesInsect Biochemistry and Molecular Biology, 2004
Wolbachia are maternally inherited bacteria that induce cytoplasmic incompatibility in mosquitoes, and are able to use these patterns of sterility to spread themselves through populations. For this reason they have been proposed as a gene drive system for mosquito genetic replacement, as well as for the reduction of population size or for modulating ...
openaire   +2 more sources

ZNRD2 Mediated Nucleoprotein Aggregation Impairs Respiratory Syncytial Virus Replication

open access: yesAdvanced Science, EarlyView.
During RSV infection, nucleoprotein (N) forms RNA‐bound oligomers. The host protein ZNRD2 binds to these oligomers, promoting their transition into insoluble aggregates. These aggregates simultaneously sequester functional N to restrict viral production and disrupt chaperonin assembly quality control by interfering with ZNRD2's role as an adaptor ...
Haiwu Zhou   +8 more
wiley   +1 more source

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