Results 111 to 120 of about 15,537 (248)
Background: Oral mucosal lesions in pemphigus vulgaris may precede cutaneous lesions and can cause diagnostic confusion. Diagnosis can be made by histopathology, direct immunofluorescence (DIF), and indirect immunofluorescence (IIF).
Amrithaa Muralitharan +2 more
doaj +1 more source
Aire-Dependent Thymic Expression of Desmoglein 3, the Autoantigen in Pemphigus Vulgaris, and Its Role in T-Cell Tolerance [PDF]
Naoko Wada +8 more
openalex +1 more source
Persistence of Anti-Desmoglein 3 IgG+ B-Cell Clones in Pemphigus Patients over Years [PDF]
Christoph M. Hammers +6 more
openalex +1 more source
Role of Desmoglein Autoantibody in the Diagnosis of Pemphigus Vulgaris: A Case Report
Pemphigus vulgaris (PV) is a group of autoimmune diseases that cause abnormalities in the form of lesions or blisters on the skin and mucous membranes. It often presents diagnostic challenges due to its varied clinical manifestations. Accurate diagnosis
Diane Lukito Setiawan +2 more
doaj
LB1723 Knocking down KLK5 by base editing restored desmoglein I expression in Netherton syndrome keratinocytes [PDF]
Vignesh Jayarajan +4 more
openalex +1 more source
The discovery of the role of Bruton’s Tyrosine Kinase (BTK) in inflammation and autoimmunity has recently led to the development of BTK inhibitors for the treatment of autoimmune diseases, including pemphigus vulgaris.
Christian Ciolfi +5 more
doaj
Rituximab therapy improves recalcitrant Pemphigus vulgaris [PDF]
Pemphigus is a severe life-threatening blistering disease associated with autoantibodies against cell adhesion proteins desmogleins 1 and 3. Patients with severe pemphigus commonly show high rates of relapse after conventional immunosuppressive therapy ...
Balighi, Kamran +7 more
core
Inflammation-induced desmoglein-2 ectodomain shedding compromises the mucosal barrier
Ryuta Kamekura +7 more
openalex +1 more source
Desmoglein 2 modulates extracellular vesicle release from squamous cell carcinoma keratinocytes
Andrew M. Overmiller +11 more
openalex +2 more sources

