Results 41 to 50 of about 1,945 (206)

Congenital Dysfibrinogenemia Presented with Massive Hematomas Formed after Hysterectomy [PDF]

, 2021
ArticleCase Reports in Clinical Medicine.
Nagata, Kentaro, Nakagami, Takefumi, Niwa, Kenji, Niwa, Kentaro, Okumura, Nobuo, Shimaoka, Ryuichiro, Takenaka, Motoki, Tanaka, Takuji   +7 more
core   +1 more source

Congenital Afibrinogenemia and Hypofibrinogenemia: Laboratory and Genetic Testing in Rare Bleeding Disorders with Life-Threatening Clinical Manifestations and Challenging Management

Diagnostics, 2021
Congenital fibrinogen disorders are rare pathologies of the hemostasis, comprising quantitative (afibrinogenemia, hypofibrinogenemia) and qualitative (dysfibrinogenemia and hypodysfibrinogenemia) disorders. The clinical phenotype is highly heterogeneous,
Tomas Simurda, Rosanna Asselta, Jana Zolkova, Monika Brunclikova, Miroslava Dobrotova, Zuzana Kolkova, Dusan Loderer, Ingrid Skornova, Jan Hudecek, Zora Lasabova, Jan Stasko, Peter Kubisz   +11 more
doaj   +1 more source

Bleeding related to disturbed fibrinolysis [PDF]

, 2016
The components and reactions of the fibrinolysis system are well understood. The pathway has fewer reactants and interactions than coagulation, but the generation of a complete quantitative model is complicated by the need to work at the solid‐liquid ...
Cawthern K.M., Colucci G., Dombret H., Goodpasture E.W., Janning M., Kashuk J.L., Kluft C., Lisman T., Longstaff C., Miura O., Oudijk E.J., Rick M.E., Rijken D.C.   +12 more
core   +1 more source

Multigenic forms of thrombophilia in habitual miscarige. [PDF]

Medičnì Perspektivi, 2015
Miscarriage is an actual problem of modern obstetrics. The frequency of miscarriage is 10-25% of all pregnancies, and habitual abortion occurs in 5%. Habitual miscarriage is considered as a typical multifactorial disease, being the result of expression ...
K. V. Voronin, N. V. Davidenko, T. O. Loskutova   +2 more
doaj   +2 more sources

A C-terminal amino acid substitution in the γ-chain caused by a novel heterozygous frameshift mutation (Fibrinogen Matsumoto VII) results in hypofibrinogenaemia [PDF]

, 2010
journal ...
Fujihara Noriko, Haneishi Ayumi, Ishida Fumihiro, Ito Toshiro, Okumura Nobuo, Terasawa Fumiko, YAMAUCHI Kazuyoshi   +6 more
core   +2 more sources

Laboratory Diagnosis of Dysfibrinogenemia

Archives of Pathology & Laboratory Medicine, 2002
AbstractDysfibrinogenemia is a coagulation disorder caused by a variety of structural abnormalities in the fibrinogen molecule that result in abnormal fibrinogen function. It can be inherited or acquired. The inherited form is associated with increased risk of bleeding, thrombosis, or both in the same patient or family. Traditionally, dysfibrinogenemia
Mark T, Cunningham, John T, Brandt, Michael, Laposata, John D, Olson   +3 more
openaire   +2 more sources

A family study of congenital dysfibrinogenemia caused by a novel mutation in the FGA gene: A case report

Open Medicine, 2020
Congenital dysfibrinogenemia (CD) is a rare hereditary fibrinogen disorder characterized by normal fibrinogen antigen levels associated with lower functional activities.
Qiao Yingli, Zhang Qisi, Xu Poshi, Deng Yuhui   +3 more
doaj   +1 more source

Biophysical Mechanisms Mediating Fibrin Fiber Lysis [PDF]

, 2017
The formation and dissolution of blood clots is both a biochemical and a biomechanical process. While much of the chemistry has been worked out for both processes, the influence of biophysical properties is less well understood. This review considers the
Hudson, Nathan E.
core   +2 more sources

Congenital dysfibrinogenemia as a rare cause of recurrent gastrointestinal bleeding [PDF]

, 2020
Introduction: Gastrointestinal bleeding is a common disease that surgeons encounter in everyday clinical practice. It is most often easy to diagnose and treat.
Kenig, Jakub, Lebowa, Weronika, Zdziarska, Joanna   +2 more
core   +1 more source

Dysfibrinogenemia Associated with Liver Disease [PDF]

Journal of Clinical Investigation, 1977
To test the possibility that a functionally abnormal fibrinogen may exist in some patients with liver disease, we studied the plasma and purified fibrinogens of five patients whose plasma thrombin times were prolonged at least 40% over normal controls. In no patient was there evidence of disseminated intravascular coagulation and/or fibrinolysis.
J E, Palascak, J, Martinez
openaire   +2 more sources