Results 31 to 40 of about 921,216 (339)

Cadherin and Integrin Regulation of Epithelial Cell Migration [PDF]

open access: yesLangmuir, 2009
These studies quantified the relative effects of E-cadherin expression and homophilic ligation on the integrin-mediated motility of epithelial cells. Micropatterned proteins were used to quantitatively titrate the ligation of E-cadherin and integrin receptors in order to assess their coordinate influence on the migration velocities of MDA-MB-231 breast
Jonathan, Silvestre   +2 more
openaire   +2 more sources

Dynamic interactions between E-cadherin and Ankyrin-G mediate epithelial cell polarity maintenance

open access: yesNature Communications, 2023
E-cadherin is an essential cell‒cell adhesion protein that mediates canonical cadherin-catenin complex formation in epithelial lateral membranes.
Chao Kong   +11 more
doaj   +1 more source

Als3 is a Candida albicans invasin that binds to cadherins and induces endocytosis by host cells. [PDF]

open access: yesPLoS Biology, 2007
Candida albicans is the most common cause of hematogenously disseminated and oropharyngeal candidiasis. Both of these diseases are characterized by fungal invasion of host cells. Previously, we have found that C.
Quynh T Phan   +8 more
doaj   +1 more source

Actin at cell-cell junctions is composed of two dynamic and functional populations [PDF]

open access: yes, 2005
The ability of epithelial cells to polarize requires cell-cell adhesion mediated by cadherin receptors. During cell-cell contact, the mechanism via which a flat, spread cell shape is changed into a tall, cuboidal epithelial morphology is not known.
Bailly, M.   +6 more
core   +1 more source

Reduced surface expression of epithelial E-cadherin evoked by interferon-gamma is Fyn kinase-dependent. [PDF]

open access: yesPLoS ONE, 2012
Interferon gamma (IFNγ) is an important regulatory cytokine that can exert a pro-inflammatory effect in the gut, where it has been shown to increase epithelial permeability via disruption of the tight junctions.
David Smyth   +3 more
doaj   +1 more source

Phosphatase Regenerating Liver3 and ECadherin Expression in Epithelial Ovarian Cancer

open access: yesIndonesian Journal of Obstetrics and Gynecology, 2016
Objective: To determine the expression of Phosphatase Regenerating Liver-3 (PRL-3) and E-Cadherin in the epithelial ovarian cancer on various stages and differentiation grades. Method: This was a cross-sectional study design conducted at Obstetrics
Risma Maharani   +2 more
doaj   +1 more source

Sphingomyelin metabolism is involved in the differentiation of MDCK cells induced by environmental hypertonicity [PDF]

open access: yes, 2015
Sphingolipids (SLs) are relevant lipid components of eukaryotic cells. Besides regulating various cellular processes, SLs provide the structural framework for plasma membrane organization.
Favale, Nicolas Octavio   +4 more
core   +1 more source

Mutations in the Epithelial Cadherin-p120-Catenin Complex Cause Mendelian Non-Syndromic Cleft Lip with or without Cleft Palate.

open access: yesAmerican Journal of Human Genetics, 2018
Non-syndromic cleft lip with or without cleft palate (NS-CL/P) is one of the most common human birth defects and is generally considered a complex trait.
L. Cox   +32 more
semanticscholar   +1 more source

Molecular mechanisms controlling the phenotype and the EMT/MET dynamics of hepatocyte [PDF]

open access: yes, 2014
The complex spatial and paracrine relationships between the various liver histotypes are essential for proper functioning of the hepatic parenchymal cells.
Fausto N   +6 more
core   +3 more sources

Extracellular cleavage of E-cadherin promotes epithelial cell extrusion [PDF]

open access: yesJournal of Cell Science, 2014
Epithelial cell extrusion and subsequent apoptosis is a key mechanism to prevent accumulation of excess cells. Conversely, when driven by oncogene expression, apical cell extrusion is followed by proliferation and represents an initial step of tumorigenesis.
Grieve, Adam G, Rabouille, Catherine
openaire   +3 more sources

Home - About - Disclaimer - Privacy