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Alterations in the Na<sup>+</sup>/H<sup>+</sup> Exchanger NHE6 and Glutamate Transporters may Influence Purkinje Cell Fate in ARSACS. [PDF]

open access: yesCerebellum
Masson LC   +6 more
europepmc   +1 more source

The shifting landscape of the preterm brain. [PDF]

open access: yesNeuron
Kratimenos P   +4 more
europepmc   +1 more source

Alternative excitotoxic hypotheses [PDF]

open access: possibleNeurology, 1992
The concept of excitotoxicity, neuronal death produced by overstimulation of excitatory amino acid receptors, has become a popular way of explaining the pathogenesis of neuronal death in a variety of acute and chronic neurologic diseases. While there is strong evidence supporting the role of excitotoxicity in acute processes such as hypoxia/ischemia ...
J. T. Greenamyre, Roger L. Albin
openaire   +2 more sources
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Excitotoxicity and mitochondria

Biochemical Society Symposia, 1999
Excitotoxicity is the process whereby a massive glutamate release in the central nervous system in response to ischaemia or related trauma leads to the delayed, predominantly necrotic death of neurons. Excitotoxicity is also implicated in a variety of slow neurodegenerative disorders.
Samantha L. Budd   +3 more
openaire   +3 more sources

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