Results 21 to 30 of about 499,256 (363)
Physicochemical Investigation of Some Oil Emulsions Oxidized by UV-B Radiation
Turkish Journal of Agriculture: Food Science and Technology, 2022 In this study, the effects of UV B (50 µW/cm2) radiation at 306 nm on oil-in-water emulsions (O/W) using canola oil, soybean oil and linoleic acid were investigated.
Temel Kan Bakır, Hüseyin Kastrati
doaj +1 more source
Increase of geometrical and positional fatty acid isomers in dark meat from broilers fed heated oils [PDF]
, 2005 Oxidation of polyunsaturated fatty acids leads to primary and secondary oxidation products. Compounds and amounts of these products vary, depending on the oxidative conditions. As these oxidation products have different absorption and biological effects,
Baucells Sánchez, María Dolores +4 more
core +2 more sources
2-pentanone production from hexanoic acid by Penicillium roqueforti from blue cheese:is this the pathway used in humans? [PDF]
, 2014 Production of 2-pentanone, a methylketone, is increased in fasting ketotic humans. Its origin is unknown. We hypothesised that it is formed via β-oxidation of hexanoic acid by the peroxisomal pathway proposed for methylketone-producing fungi and yeasts ...
Mills, Graham A., Walker, Valerie
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Metabolites, 2021 In the presence of high abundance of exogenous fatty acids, cells either store fatty acids in lipid droplets or oxidize them in mitochondria. In this study, we aimed to explore a novel and direct role of mitochondrial fission in lipid homeostasis in HeLa
Jae-Eun Song +8 more
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Medium-chain acyl-CoA dehydrogenase deficiency in gene-targeted mice [PDF]
, 2005 Medium-chain acyl-CoA dehydrogenase (MCAD) deficiency is the most common inherited disorder of mitochondrial fatty acid β-oxidation in humans. To better understand the pathogenesis of this disease, we developed a mouse model for MCAD deficiency (MCAD-/-)
David Valle +9 more
core +4 more sources
Frontiers in Pharmacology, 2021 Fatty acid β-oxidation is an essential pathogenic mechanism in nonalcoholic fatty liver disease (NAFLD), and TATA-box binding protein associated factor 9 (TAF9) has been reported to be involved in the regulation of fatty acid β-oxidation.
Ruiwen Wang +10 more
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Stable isotope metabolomics of pulmonary artery smooth muscle and endothelial cells in pulmonary hypertension and with TGF-beta treatment. [PDF]
, 2020 Altered metabolism in pulmonary artery smooth muscle cells (PASMCs) and endothelial cells (PAECs) contributes to the pathology of pulmonary hypertension (PH), but changes in substrate uptake and how substrates are utilized have not been fully ...
D' Alessandro, Angelo +12 more
core +1 more source
Prenatal hyperandrogenism induces alterations that affect liver lipid metabolism [PDF]
, 2016 Prenatal hyperandrogenism is hypothesized as one of the main factors contributing to26 the development of polycystic ovary syndrome (PCOS). PCOS patients have high risk27 of developing fatty liver and steatosis.
Abruzzese, Giselle Adriana +6 more
core +2 more sources
Alteration of Energy Substrates and ROS Production in Diabetic Cardiomyopathy
Mediators of Inflammation, 2013 Diabetic cardiomyopathy is initiated by alterations in energy substrates. Despite excess of plasma glucose and lipids, the diabetic heart almost exclusively depends on fatty acid degradation. Glycolytic enzymes and transporters are impaired by fatty acid
O. Lorenzo +4 more
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Cell Reports, 2017 The liver has a large capacity for mitochondrial fatty acid β-oxidation, which is critical for systemic metabolic adaptations such as gluconeogenesis and ketogenesis.
Jieun Lee +7 more
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