Results 241 to 250 of about 147,915 (338)
Ferroptosis in Neonatal Hypoxic-Ischemic Encephalopathy: Mechanisms and the Therapeutic Potential of Vitamin D/VDR Signaling. [PDF]
Cai Y, Zhou W.
europepmc +1 more source
Activation of GSDME by all- trans -retinal increases sensitivity to photoreceptor ferroptosis [PDF]
Bo Yang +7 more
openalex +1 more source
Restoration of the mitochondrial Fe–S protein FDX1 suppresses clear cell renal cell carcinoma by coupling ferroptosis with tumor‐intrinsic innate immune activation. FDX1 induces mitochondrial dysfunction and mtDNA/mt‐dsRNA release, activating cGAS–STING and RIG‐I/MDA5–MAVS signaling to elicit type I interferon responses, thereby promoting antigen ...
Xing Huang +13 more
wiley +1 more source
CCNE1 Exerts a Protective Effect on Parkinson's Disease by Regulating Ferroptosis-Related Proteins. [PDF]
Fu J +5 more
europepmc +1 more source
The Immune Microenvironment in Liver Cancer: From Analysis to Targeting
This review explores how smart nanomedicines overcome the immunosuppressive tumor microenvironment in liver cancer. It details targeted delivery strategies and immune reprogramming mechanisms, including remodeling abnormal physiology, modulating metabolism, and inducing immunogenic cell death. The article highlights the paradigm shift toward multimodal
Jiaming Lan +5 more
wiley +1 more source
Harnessing the gut microbiome to modulate ferroptosis: a metabolic strategy for the treatment of digestive tract cancers. [PDF]
Wen J +10 more
europepmc +1 more source
BIOM-28. FOSL2 PROMOTES FERROPTOSIS RESISTANCE IN GLIOBLASTOMA [PDF]
Yanli Tan +5 more
openalex +1 more source
Advanced Therapeutic Approach Based on LDHs–Mimetic Oxidoreductase
Overview of Sructure, Multienzyme‐mimicking Activity and Advanced Therapeutic Approach Based on LDHs–Mimetic Oxidoreductase. Abstract Redox dysregulation is recognized as a key driver in the pathophysiology of numerous refractory diseases, contributing significantly to the progression and poor prognosis.
Kai Song +9 more
wiley +1 more source

