Fibroblast Growth Factor-23 (FGF-23) e Sindrome Cardiorenale
Giornale di Clinica Nefrologia e Dialisi, 2015 non disponibile (Cardionephrology)
Luca Di Lullo +6 more
doaj +1 more source
Serum intact fibroblast growth factor 23 in healthy paediatric population
Open Medicine, 2021 It is believed that fibroblast growth factor 23 (FGF23) can become an early biomarker of chronic kidney disease progression. Data on FGF23 age dependency are inconsistent.
Stanczyk Malgorzata +3 more
doaj +1 more source
Comparison of particle-size analyzing laboratory methods [PDF]
, 2015 Patients with chronic renal failure are known to have renal osteodystrophy (bone disease) and increased calcification of vessels. A new marker of bone disease, sclerostin, the two pro-inflammatory cytokines tumour necrosis factor-alpha (TNF-alpha) and ...
Barta, Károly +6 more
core +1 more source
MicroRNA-297a regulates vascular calcification by targeting fibroblast growth factor 23 [PDF]
Iranian Journal of Basic Medical Sciences, 2016 Objective(s): Vascular calcification is one the major characteristics in patients with various types of chronic inflammatory disorders. MiRNAs have been shown to be involved in many normal biological functions as well as diseases; however, their role in ...
Shouhua Zheng +6 more
doaj +1 more source
Intact fibroblast growth factor 23 and fragments in plasma from Gambian children [PDF]
, 2012 SUMMARY: Fibroblast growth factor 23 (FGF23) is grossly elevated in Gambian children with rickets and, at a lower prevalence, in those without bone deformities.
A. Prentice +2 more
core +1 more source
Fibroblast Growth Factor 23 and Klotho in AKI [PDF]
Seminars in Nephrology, 2019 Acute kidney injury (AKI) is associated with many of the same mineral metabolite abnormalities that are observed in chronic kidney disease. These include increased circulating levels of the osteocyte-derived, vitamin D-regulating hormone, fibroblast growth factor 23 (FGF23), and decreased renal expression of klotho, the co-receptor for FGF23.
Marta, Christov +3 more
openaire +2 more sources
An Unusual Combination of Neurological Manifestations and Sudden Vision Loss in a Child with Familial Hyperphosphatemic Tumoral Calcinosis [PDF]
, 2019 Hyperphosphatemia in the absence of renal failure is an unusual occurrence, particularly in children, but is a common primary feature of familial hyperphosphatemic tumor calcinosis.
Acton, Dena +7 more
core +1 more source
Iron and fibroblast growth factor 23 in X-linked hypophosphatemia [PDF]
, 2014 Background Excess fibroblast growth factor 23 (FGF23) causes hypophosphatemia in autosomal dominant hypophosphatemic rickets (ADHR) and X-linked hypophosphatemia (XLH). Iron status influences C-terminal FGF23 (incorporating fragments plus intact FGF23)
Econs, Michael J. +3 more
core +1 more source
Multiple faces of fibroblast growth factor-23 [PDF]
Current Opinion in Nephrology and Hypertension, 2016 This review examines the role of fibroblast growth factor-23 (FGF-23) in mineral metabolism, innate immunity and adverse cardiovascular outcomes.FGF-23, produced by osteocytes in bone, activates FGFR/α-Klotho (α-Kl) complexes in the kidney. The resulting bone-kidney axis coordinates renal phosphate reabsorption with bone mineralization, and creates a ...
Xiaobin, Han, L Darryl, Quarles
openaire +2 more sources
Predictors of intact and C-terminal fibroblast growth factor 23 in Gambian children [PDF]
, 2013 Elevated C-terminal fibroblast growth factor 23 (C-FGF23) concentrations have been reported in Gambian children with and without putative Ca-deficiency rickets.
Assar, Shima +4 more
core +1 more source