Results 251 to 260 of about 142,631 (286)

Exploring the Mahuang Fuzi Xixin Decoction's mechanism for treating Alzheimer's disease using molecular docking and network pharmacology. [PDF]

Front Aging Neurosci
Chen C, Shao Q, Zhou S.
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Disulfiram activates autophagy via proteasome inhibition and c-Fos/beclin-1 upregulation, synergizing with chloroquine. [PDF]

Cell Death Discov
Wang K, Wang Z, Peng W, Li G, Xiao H, Zhong Z, He Y, Yu Y, Song Y, Xiang L, Wu C.   +10 more
europepmc   +1 more source

Molecular determinants of neoadjuvant chemotherapy resistance in breast cancer: An analysis of gene expression and tumor microenvironment. [PDF]

PLoS One
Guevara-Nieto HM, Orozco-Castaño CA, Parra-Medina R, Poveda-Garavito JN, Garai J, Zabaleta J, López-Kleine L, Combita AL.   +7 more
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Sleep and wakefulness in c-fos and fos B gene knockout mice

Molecular Brain Research, 2000
G-protein coupled receptor (GPCR) stimulation has been implicated in the regulation of sleep. Upon stimulation of a GPCR an intracellular cascade involving second and third messengers is initiated. The latter include the fos-family of immediate early genes (IEGs). Although there is considerable evidence indicating that IEGs are expressed in response to
P J, Shiromani, R, Basheer, J, Thakkar, D, Wagner, M A, Greco, M E, Charness   +5 more
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c-fos antisense RNA blocks expression of c-fos gene in F9 embryonal carcinoma cells

Cell Differentiation and Development, 1988
To study the function of proto-oncogene c-fos, we prepared an antisense plasmid that expresses in mammalian cells c-fos antisense RNA which is complementary to the endogenous c-fos mRNA. Upon transfection into undifferentiated F9 EC cells, the antisense plasmid directed constitutive expression of a large amount of c-fos antisense RNA.
B Z, Levi, J W, Kasik, K, Ozato
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Alteration of c-fos gene methylation in human gliomas

Molecular Carcinogenesis, 1996
In an attempt to find a common DNA alteration occurring in human glioma, we examined DNA methylation in 34 gliomas of various pathological grades and compared them with those in normal cerebral subcortex DNA. The total methylated cytosine levels in the genome did not differ appreciably between the tumors and the normal tissues; however, the degree of ...
S, Uyeno, J, Komura, R, Tawa, Y, Aoki, M, Nata, H, Sasano, H, Nakura, K, Sagisaka, H, Sakurai, T, Kayama, T, Yoshimoto, T, Ono   +11 more
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Viral and Cellular Fos Gene

1985
A remarkable discovery of the past few years is the fact that many acutely oncogenic retroviruses contain sequences as an integral part of their genome which have homologs in normal cells (1). Acquisition of normal cellular sequences by retroviruses, usually at the expense of genes required for replication, enables them to induce neoplasias in vivo and
I. M. Verma, T. Curran, A. D. Miller, C. Van Beveren   +3 more
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Induction of c-fosGene Expression by Interferons

Journal of Interferon Research, 1988
We show that murine interferons (IFNs)-alpha/beta and -gamma induce expression of the protooncogene c-fos in F9 embryonal carcinoma cells, NIH-3T3 cells, and other tissue culture cells. The induction of c-fos mRNA is rapid and transient in that the mRNA appears within 15 min and disappears by 2 h following IFN treatment. The protein synthesis inhibitor
Y J, Wan, B Z, Levi, K, Ozato
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Expression of the c- fos Gene and of an fos -Related Gene Is Stimulated by Platelet-Derived Growth Factor

Science, 1984
Complementary DNA clones of genes induced by platelet-derived growth factor (PDGF) in BALB/c-3T3 cells were isolated; one such clone contains a domain having nucleotide sequence homology with the third exon of c- fos .
B H, Cochran, J, Zullo, I M, Verma, C D, Stiles   +3 more
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