Results 251 to 260 of about 2,017,768 (306)

[Human gene p53].

Postepy biochemii, 1994
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Disruption of macrophage migration inhibitory factor signaling induces major tumor-associated macrophage phenotypes in human M2 macrophages. [PDF]

Mol Biomed
Klaver D, Gander H, Frena B, Martin M, Amato M, Richter J, Pichler R, Thurnher M.   +7 more
europepmc   +1 more source

Icaritin improves pregnancy outcomes in endometriosis by inhibiting ovarian granulosa cell senescence. [PDF]

Mol Biol Rep
He Y, Ding J, Li Y, Sun J, Wei C, Sun N, Jiang R, Zhao Z, Liang X, Chen Y, Cheng W, Zhang L, Song D, Sun S, Yu C.   +14 more
europepmc   +1 more source

The Dynamic Alliance of p53 and Metabolism in the Tumor Microenvironment Shapes Tumor Evolution. [PDF]

Bioessays
Joruiz SM, Napoletano F, Bertolio R, Del Sal G.   +3 more
europepmc   +1 more source

SMARCA4 loss reprograms p300 chromatin occupancy to subvert p53-mediated transcriptional repression in ovarian small cell carcinoma. [PDF]

Nat Commun
Aceto G, Liu K, Arabzadeh A, Tsai S, Xue Y, Monast A, Pilon V, Han M, Morin G, Pavlakis K, Fu L, Park M, Foulkes WD, Wang Y, Huang S.   +14 more
europepmc   +1 more source

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The p53 network: p53 and its downstream genes

Colloids and Surfaces B: Biointerfaces, 2007
The tumor-suppressor gene p53 and its downstream genes consist of a complicated gene network. p53 is a key molecular node in the network, which is activated in response to several cellular signals resulting in the maintenance of genetic stability. Several cellular signals may activate the p53 network.
Kun-Xian, Shu, Biao, Li, Li-Xiang, Wu
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The p53 tumour suppressor gene

Nature, 1991
The cell cycle is composed of a series of steps which can be negatively or positively regulated by various factors. Chief among the negative regulators is the p53 protein. Alteration or inactivation of p53 by mutation, or by its interactions with oncogene products of DNA tumour viruses, can lead to cancer.
A J, Levine, J, Momand, C A, Finlay
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CLINICAL IMPLICATIONS OF THE p53 GENE

Annual Review of Medicine, 1996
▪ Abstract  The capacity for malignant growth is acquired by the stepwise accumulation of defects in specific genes regulating cell growth and tissue homeostasis. Although several hundred genes are known to control growth, molecular genetic studies in cancer show that few of these are consistently involved in the natural history of human cancer, and ...
D, Sidransky, M, Hollstein
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Gene-targeting and the p53 tumor-suppressor gene

Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1994
Gene-targeting techniques are now frequently applied to embryonic stem (ES) cells to introduce mutations of endogenous genes in mice. Modifications introduced into tumor-suppressor genes by this technology have produced mice and cell lines with unique tumorigenic and growth characteristics, respectively.
A, Sands, L A, Donehower, A, Bradley
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