Results 191 to 200 of about 45,138 (251)
Focal segmental glomerulosclerosis
Orvosi Hetilap, 2008 A focalis segmentalis glomerulosclerosis fénymikroszkópos vizsgálattal látható jellegzetes morfológiájú glomeruluskárosodás, többféle szövettani altípussal és változó klinikai képpel. A különböző szövettani variánsokat újabban rendszerezték (Columbia-klasszifikáció).
Tibor, Vas +5 more
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Focal Segmental Glomerulosclerosis [PDF]
New England Journal of Medicine, 2011 Focal segmental glomerulosclerosis accounts for approximately 20% of cases of the nephrotic syndrome in children and 40% of such cases in adults, with an estimated incidence of 7 per 1 million.(1) It is the most common primary glomerular disorder causing end-stage renal disease in the United States, with a prevalence of 4%.(2) The cardinal feature is ...
Vivette D, D'Agati +2 more
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Modulation of glomerulosclerosis
Seminars in Immunopathology, 2007Regardless of the initial injury, the long-term consequence for the patient depends upon the ensuing balance of profibrotic vs reparative modulators activated. The glomerulus has some capacity for repair. Even when sclerosis has developed with accumulation of extracellular matrix, this lesion may be remodeled, with a change in balance between ...
Li-Jun, Ma, Agnes B, Fogo
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Loss of decay-accelerating factor triggers podocyte injury and glomerulosclerosis [PDF]
Journal of Experimental Medicine, 2020 Kidney glomerulosclerosis commonly progresses to end-stage kidney failure, but pathogenic mechanisms are still poorly understood. Here, we show that podocyte expression of decay-accelerating factor (DAF/CD55), a complement C3 convertase regulator ...
Deborah Malvi +2 more
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Immunohistopathology of Diabetic Glomerulosclerosis
Diabetes, 1972The cause and nature of the lesions in small blood vessels in diabetes mellitus is still unknown. Immunological mechanisms possibly related to reactions between insulin and antibodies to insulin, or damage caused by transudation of serum proteins, have been proposed by several investigators.
N G, Westberg, A F, Michael
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The role of the podocyte in glomerulosclerosis
Current Opinion in Nephrology and Hypertension, 1999Focal segmental glomerulosclerosis is a pathological hallmark of many forms of progressive renal disease. The 'classic' lesion, based on the adhesion of the capillary tuft to Bowman's capsule, results from the loss of podocytes from the capillary basement membrane.
W, Kriz, K V, Lemley
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Glomerulosclerosis and hyalinosis in rabbits
Pathology, 1992Histological appearances of remnant kidney in female New Zealand white rabbits undergoing left nephrectomy at 6 mths were studied. All 20 rabbits had evidence of previous Encephalitozoon cuniculi (E. cuniculi) infection. Half of the 10 uninephrectomized and 10 control animals completed 3 pregnancies before sacrifice (15 mths).
D K, Packham +3 more
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The Irish Journal of Medical Science, 1950
1. An analysis of a series of authenticated, autopsied cases of diabetes mellitus has confirmed the specificity of nodular glomerulosclerosis and enlarged the interpretation to include the hyalinization of the efferent arteriole.
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1. An analysis of a series of authenticated, autopsied cases of diabetes mellitus has confirmed the specificity of nodular glomerulosclerosis and enlarged the interpretation to include the hyalinization of the efferent arteriole.
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The Evolution of Intercapillary Glomerulosclerosis
New England Journal of Medicine, 1949IN 1936, Kimmelstiel and Wilson1 described a diffuse, microscopic lesion in the kidneys of 8 middle-aged and elderly diabetic patients with hypertension, profuse albuminuria and generalized edema. The abnormality was termed "intercapillary glomerulosclerosis" and consisted of striking changes in the intercapillary tissue characterized by large hyaline ...
H A, DEROW, M J, SCHLESINGER
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Pathogenesis of Focal Glomerulosclerosis
Nephron, 2001Focal segmental glomerulosclerosis (FSGS) is the histologic expression of diverse processes affecting the renal glomeruli and occurring in primary and secondary forms. A number of pathogenic factors have been identified in primary FSGS, and multiple etiologies have been defined as contributing factors for the development of secondary FSGS.
W K, Bolton, E, Abdel-Rahman
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