Results 161 to 170 of about 291,048 (366)
ON THE MECHANISM OF PROTECTION OF ENDOTOXIN SHOCK BY GLUCOCORTICOIDS
Tokuro FUKUDA+2 more
openalex +2 more sources
INHIBITION OF GROWTH AND DNA SYNTHESIS OF CELLS FROM AN ALVEOLAR CELL CARCINOMA BY GLUCOCORTICOIDS [PDF]
Kenneth L. Jones+2 more
openalex +1 more source
Objective Podocytes are integral to the maintenance of the glomerular filtration barrier. Their injury results in proteinuria and disease progression in lupus nephritis (LN). Aberrant IgG glycosylation drives podocyte injury in LN and leads to cytoskeletal rearrangement, motility changes, and decreased nephrin production.
Rhea Bhargava+9 more
wiley +1 more source
Objective This study characterized the impact of voclosporin on kidney histology in patients with lupus nephritis (LN) who had protocolized repeat kidney biopsies in the AURORA clinical trials. Methods Patients were randomized to voclosporin or placebo treatment for up to 3 years; all patients received mycophenolate mofetil and low‐dose glucocorticoids.
Brad H. Rovin+9 more
wiley +1 more source
INHIBITORY EFFECT OF ADRENAL GLUCOCORTICOIDS ON HISTAMINE RELEASE
Hidemasa Yamasaki, Takashi Yamamoto
openalex +1 more source
Gout and NLRP3 Inflammasome Biology
This review describes the three broad stages of acute inflammation in the context of gout: initiation, leucocyte mobilization, and self‐resolution. A typical case of a gout flare is presented. The role of the NLRP3 inflammasome in acute monosodium urate crystal–induced inflammation is reviewed in detail.
Raewyn Poulsen, Nicola Dalbeth
wiley +1 more source
Abstract Background and Aims Reliable noninvasive biomarkers are an unmet clinical need for the diagnosis of NASH. This study investigates the diagnostic accuracy of the circulating triggering receptor expressed on myeloid cells 2 (plasma TREM2) as a biomarker for NASH in patients with NAFLD and elevated liver stiffness.
Vineesh Indira Chandran+17 more
wiley +1 more source
Effect of Glucocorticoids on Thyroid Function of Cattle
C. W. Turner+2 more
openalex +1 more source
Objective Systemic juvenile idiopathic arthritis (sJIA) is a chronic childhood disease classically attributed to innate immune cell dysregulation. This study aimed to elucidate the role of innate lymphoid cells (ILCs), including natural killer (NK) cells and helper‐ILCs (hILCs), in sJIA during clinically inactive disease (CID) through phenotypic and ...
Linda Quatrini+19 more
wiley +1 more source