Results 211 to 220 of about 64,826 (258)
Colchicine Inhibits Chemokine Production in Monosodium Urate-Stimulated Innate Immune Cells. [PDF]
Merlo Pich LM +5 more
europepmc +1 more source
A Bidirectional Analysis of Inflammatory Bowel Disease and Gout: Epidemiologic Evidence of a Stronger Association in Crohn's Disease from a Nationwide Cohort Study in South Korea. [PDF]
Yoo DM +7 more
europepmc +1 more source
Serum uric acid and the trajectory of cardiometabolic multimorbidity: a prospective study conducted with data from the UK Biobank. [PDF]
Wu J +6 more
europepmc +1 more source
Transformer-Based Multiomics Study Identifies Important Role of Glycine, Serine, and Threonine Metabolism Pathway in Rheumatoid Arthritis Complicated by Anemia. [PDF]
Huang J +16 more
europepmc +1 more source
The treat to target strategy looks attractive for management of gout because it removes the causal mechanism inducing formation, growth, and aggregation of urate crystals. Further reduction of serum urate (sUA) levels below the threshold may dissolve crystals more rapidly. It is generally agreed that sUA less than 6 mg/dL is acceptable as a therapeutic
Yusuke Kawamura +2 more
exaly +10 more sources
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New England Journal of Medicine, 2011
This issue provides a clinical overview of gout, focusing on prevention and screening, diagnosis, and treatment. The content of In the Clinic is drawn from the clinical information and education resources of the American College of Physicians (ACP), including MKSAP (Medical Knowledge and Self-Assessment Program).
Franz H, Messerli +2 more
openaire +4 more sources
This issue provides a clinical overview of gout, focusing on prevention and screening, diagnosis, and treatment. The content of In the Clinic is drawn from the clinical information and education resources of the American College of Physicians (ACP), including MKSAP (Medical Knowledge and Self-Assessment Program).
Franz H, Messerli +2 more
openaire +4 more sources
New England Journal of Medicine, 2004
We have reviewed the latest publications on epidemiology of gout; also there have been new insights into the regulation of the inflammation resulting from the regular interaction occurring between MSU crystals and cells in both asymptomatic and symptomatic gouty joints. Finally we review different publications of clinical interest.The incidence of gout
Alastair L, Hepburn, Michael D, Feher
openaire +4 more sources
We have reviewed the latest publications on epidemiology of gout; also there have been new insights into the regulation of the inflammation resulting from the regular interaction occurring between MSU crystals and cells in both asymptomatic and symptomatic gouty joints. Finally we review different publications of clinical interest.The incidence of gout
Alastair L, Hepburn, Michael D, Feher
openaire +4 more sources
BMJ, 2010
Gout is a common arthritis caused by deposition of monosodium urate crystals within joints after chronic hyperuricaemia. It affects 1-2% of adults in developed countries, where it is the most common inflammatory arthritis in men. Epidemiological data are consistent with a rise in prevalence of gout.
Pascal, Richette, Thomas, Bardin
openaire +4 more sources
Gout is a common arthritis caused by deposition of monosodium urate crystals within joints after chronic hyperuricaemia. It affects 1-2% of adults in developed countries, where it is the most common inflammatory arthritis in men. Epidemiological data are consistent with a rise in prevalence of gout.
Pascal, Richette, Thomas, Bardin
openaire +4 more sources
The Lancet, 2016
Gout is a chronic disease of deposition of monosodium urate crystals, which form in the presence of increased urate concentrations. Although environmental factors contribute to hyperuricaemia, renal and gut excretion of urate is central to regulation of serum urate, and genetic factors are important.
Dalbeth, Nicola, Merriman, TR, Stamp, LK
openaire +3 more sources
Gout is a chronic disease of deposition of monosodium urate crystals, which form in the presence of increased urate concentrations. Although environmental factors contribute to hyperuricaemia, renal and gut excretion of urate is central to regulation of serum urate, and genetic factors are important.
Dalbeth, Nicola, Merriman, TR, Stamp, LK
openaire +3 more sources

