Results 251 to 260 of about 194,359 (349)
Literature review analysis of aortitis induced by granulocyte-colony stimulating factor. [PDF]
Zhao T, Xu H.
europepmc +1 more source
In this Turkish cohort, several risk indicators were identified for peri‐implantitis and peri‐implant soft‐tissue dehiscence. Several PICF immunological markers, particularly IL‐8 and TNF‐α, showed promising diagnostic potential for peri‐implantitis.
Sila Cagri Isler +5 more
wiley +1 more source
Granulocyte colony-stimulating factor combined with SOFA score for mortality prediction in patients with sepsis. [PDF]
Fu X, Zhang Y, Wang J, Liu Y, Wei B.
europepmc +1 more source
Systemic, Lifestyle and Environmental Modifying Factors in the Pathogenesis of Periodontitis
A variety of impacting factors in the pathogenesis of periodontitis exist, including systemic, lifestyle, and environmental factors. This review highlights the manifold mechanistic aspects of the link between the pathogenesis of periodontitis, addressing these numerous factors to supplement the long‐standing knowledge of the progression of the disease.
Groeger Sabine Elisabeth +5 more
wiley +1 more source
Elevation of Granulocyte Colony Stimulating Factor in Human AMD Donor RPE-Choroid. [PDF]
Mulfaul K +10 more
europepmc +1 more source
A graphical abstract recapping the different sources of dental, periodontal, and other oral‐derived mesenchymal stromal cells (MSCs) and their regenerative mechanisms and potentials. The review's article findings bridge fundamental biological science with translational advances, highlighting the significance of MSCs in craniofacial regenerative ...
Karim M. Fawzy El‐Sayed +6 more
wiley +1 more source
Granulocyte colony-stimulating factor induced T-cell hyporesponsiveness via modulation of CD177<sup>+</sup>S100A<sup>hi</sup> neutrophils in unexplained recurrent pregnancy loss. [PDF]
Li PF, Zhang X, Zheng PS.
europepmc +1 more source
Inflammatory and Immunological Basis of Periodontal Diseases
The periodontal lesion emerges as an evolving immunological battlefield, where host–microbiome interactions, dysregulated immune responses, fragile resolution mechanisms, and inflammophilic dysbiosis converge to shift the balance from homeostasis to unrestrained tissue destruction.
Giacomo Baima +3 more
wiley +1 more source

