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Microchimerism in Graves’ Disease [PDF]

Journal of Thyroid Research, 2012
Microchimerism is the presence of cells from one individual in another genetically distinct individual. Pregnancy is the main cause of natural microchimerism through transplacental bidirectional cell trafficking between mother and ...
Galofre, J.C. (Juan Carlos)
core   +6 more sources

Graves’ disease [PDF]

Nature Reviews Disease Primers, 2020
Graves' disease (GD) is an autoimmune disease that primarily affects the thyroid gland. It is the most common cause of hyperthyroidism and occurs at all ages but especially in women of reproductive age. Graves' hyperthyroidism is caused by autoantibodies to the thyroid-stimulating hormone receptor (TSHR) that act as agonists and induce excessive ...
Davies, Terry F., Andersen, Stig, Latif, Rauf, Nagayama, Yuji, Barbesino, Giuseppe, Brito, Maria, Eckstein, Anja, Stagnaro-Green, Alex, Kahaly, George J.   +8 more
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Thyroid function disorders [PDF]

Arhiv za farmaciju, 2022
Thyroid function disorders are the most common endocrine disorders in humans. They are frequently diagnosed, with prevalence varying between 2 and 6%, and a female to male ratio of up to 10:1.
Stojković Mirjana
doaj   +1 more source

What kind of therapeutic solutions for Graves’ disease does modern medicine propose? - a review of the literature

Journal of Education, Health and Sport, 2023
Background: Graves' disease is responsible for 60-80% causes of hyperthyreosis. An important role in the pathogenesis is played by anti-TSHR immunoglobulins (TRAb), that stimulate TSH receptors leading to excessive hormone synthesis and release by the ...
Natalia Woś, Karolina Szala-Czerwonka, Adrian Rejmer, Katarzyna Rojek, Lucjan Bednarz, Karolina Wijas, Kinga Bialic, Rafał Bakalarczyk, Artur Bialic, Paweł Majewski   +9 more
doaj   +1 more source

Evans syndrome and Graves' Disease: An unusual presentation – A case report

Apollo Medicine, 2022
Introduction: Evans syndrome (ES), an infrequently encountered haematological condition, is characterized by coombs-positive autoimmune hemolytic anaemia (AIHA) in association with immune thrombocytopenia (ITP). The association of ES with Graves' disease
Amtoj Singh Lamba, Monica Gupta, Samiksha Gupta, Rayidi Rajesh   +3 more
doaj   +1 more source

P-082 CHALLENGES IN PATIENT CARE WITH CUSHING DISEASE AND COMORBIDITIES (PRIMARY HYPERPARATHYROIDISM, GRAVES’ DISEASE) [PDF]

JCEM Case Rep
Nataliia Budul, Irena Ilovayskaya, Irina Komerdus   +2 more
europepmc   +2 more sources

Correction to: "Impact of Minimal Dose Strategy Before Antithyroid Drug Discontinuation on Relapse Risk in Graves' Disease".

J Clin Endocrinol Metab
europepmc   +2 more sources

Graves-Basedow disease and potential risk factors [PDF]

Vojnosanitetski Pregled, 2008
Background/Aim. Graves-Basedow disease is a common multifactorial genetic syndrome, which is determined by several genes and environmental factors. The aim of the present study was to investigate the presence of risk factors for developing Graves-Basedow
Živanović Dušica, Šipetić Sandra, Stamenković-Radak Marina, Milašin Jelena   +3 more
doaj   +1 more source

A Case of Thyrotoxic Periodic Paralysis and Graves’ Ophthalmopathy Patient in Coincidence with Chronic Hepatitis B Infection

The Indonesian Journal of Gastroenterology, Hepatology and Digestive Endoscopy, 2022
Graves' disease is an autoimmune thyroid disease with several characteristic symptoms and signs. Graves' ophthalmopathy, an inflammatory disease in the orbital area, is the main extrathyroid manifestation of Graves' disease.
Herry Krisnata Ginting, Soebagijo Adi
doaj   +1 more source

Pretibial myxedema: Paradoxical manifestation of thyroid dermopathy after I-131 ablation therapy

Journal of Medical Sciences, 2020
Pretibial myxedema (PM) is an unusual extrathyroidal manifestation of Graves' disease (GD), which affects approximately 1%–5% of the patients. In addition, PM usually occurs in the hyperthyroid state of GD, and the high level of thyroid autoantibodies is
Bing-Sian Lin, Yi-Hsien Chen, Chih-Tsung Hung   +2 more
doaj   +1 more source