Results 11 to 20 of about 9,143 (202)

ROLE OF HUMAN PAPILLOMAVIRUS GENOTYPE 16 IN PATHOLOGY OF CERVICAL CANCER

open access: yesŽurnal Grodnenskogo Gosudarstvennogo Medicinskogo Universiteta, 2022
Cervical cancer is the fourth most common malignant neoplasm in women worldwide and the third in Iraq. One of the reasons accounting for this is the human papillomavirus (HPV), which is found in 70% of Iraqis.
A. T. Jalil, A. E. Karevskiy
doaj   +1 more source

Study of HPV16 L1 Capsid Seropositivity in Patients with Oral and Oropharyngeal Cancers

open access: yesJournal of Orofacial Sciences, 2021
Introduction: Epidemiologic studies show that incidence of oral and oropharyngeal cancers (OPCs) is increasing worldwide over the last decades and the increase is more marked in men than in women.
Shalini Kanaparti   +5 more
doaj   +1 more source

Frequency of Human Papilloma Virus 16/18 Patients With Prostate Cancer by Polymerase Chain Reaction Method in Hospitals of Tabriz City, Iran

open access: yesThe Journal of Qazvin University of Medical Sciences, 2022
Background: Prostate cancer (PCa), as the fifth leading cause of death, is the second most common cancer diagnosis in men worldwide. Human papillomavirus (HPV) can potentially contribute to PCa development and chronic inflammation. HPV infection leads to
Robabeh Faghany Baladehi   +7 more
doaj   +1 more source

Role of HPV16 E1 in cervical carcinogenesis [PDF]

open access: yesFrontiers in Cellular and Infection Microbiology, 2022
Cervical cancer is the fourth most common cancer in women worldwide. More than 90% of cases are caused by the human papillomavirus (HPV). Vaccines developed only guard against a few HPV types and do not protect people who have already been infected.
Fern Baedyananda   +8 more
openaire   +3 more sources

The absence of high-risk human papillomavirus in Czech non-small cell lung cancer cases

open access: yesBiomedical Papers, 2020
Aims: The purpose of our study was to examine the presence of human papillomavirus (HPV) DNA in Czech patients with non-small cell lung cancer (NSCLC).
Hana Jaworek   +4 more
doaj   +1 more source

Long-term follow-up of HPV16-positive women: persistence of the same genetic variant and low prevalence of variant co-infections. [PDF]

open access: yesPLoS ONE, 2013
HPV16 variants correlate with geographic origin and ethnicity. The association between infection with a specific variant and the cervical disease risk remains unclear.
Daan T Geraets   +5 more
doaj   +1 more source

HPV16-specific Region Capture Hi-C determines definitive HPV16 integration sites.

open access: yes, 2021
CIRCOS plots show sequence interactions between HPV16 (orange) and host chromosomes (various) for clones (A) G2, (B) D2, (C) H, (D) F and (E) A5.
Emma L. A. Drane (11331464)   +11 more
core   +1 more source

Anti-L1 antibody-bound HPV16 pseudovirus is degraded intracellularly via TRIM21/proteasomal pathway

open access: yesVirology Journal, 2022
Background Persistent HPV16 infection is the leading risk factor for developing cervical cancer. Anti-L1 antibodies against HPV16 produced in HPV16 infections play diverse roles in the clearance of virus infection and prevention of persistence.
Meiying Li   +5 more
doaj   +1 more source

The analysis of L1 gene variability of Human papilloma virus type 16 in our population [PDF]

open access: yesMedicinski Podmladak, 2022
Introduction: Human papilloma viruses (HPV) have been identified as a major etiological factor in the pathogenesis of cervical cancer. High-risk type HPV16 has the greatest medical significance. Based on differences in the nucleotide sequence of the type
Milošević Filip   +2 more
doaj  

HPV16-host breakpoints identified in clones H, F and A5 by HPV16-specific Region Capture Hi-C.

open access: yes, 2021
(A) Capture Hi-C data is presented 515 kbp across the HPV16 integration locus for W12 clone H. The 5’ and 3’ breakpoints of the virus are indicated by the tallest red bars and are labelled with black arrowheads, running leftward due to the direction of ...
Emma L. A. Drane (11331464)   +11 more
core   +1 more source

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