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Signaling in Induced Resistance

2010
Induced mechanisms are by definition imperceptible or less active in uninfected, unstressed, or untreated plants, but can be activated by pathogen infection, stress, or chemical treatment to inhibit the replication and movement of virus in the host. In contrast, defenses that are pre-existing or serve to limit virus propagation and spread in otherwise ...
John P, Carr   +2 more
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Induced Resistance in Legumes

1995
Evaluates the evidence for the occurrence and underlying mechanisms in bean Phaseolus spp. where most has been done, then assesses evidence in soybean Glycine max, pea Pisum sativum and other legumes. A greal deal of recent research has been carried out in legumes on molecular bases of expression and elicitation of resistance to avirulent strains of ...
Deverall, B. J., Dann, E. K.
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Induced Resistance to Disease

2018
Infection of one leaf of a plant with virulent or avirulent pathogens that cause localized necrotic lesions often results in physiological and biochemical changes in the plant that enhance the resistance of the plant to further infection. This type of response, which can be either very localized or manifested throughout the plant (systemic), is ...
Hammerschmidt, R., Dann, E. K.
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Unraveling mycorrhiza-induced resistance

Current Opinion in Plant Biology, 2007
Arbuscular mycorrhizal symbioses have a significant impact on plant interactions with other organisms. Increased resistance to soil-borne pathogens has been widely described in mycorrhizal plants. By contrast, effects on shoot diseases largely rely on the lifestyle and challenge strategy of the attacker.
María J, Pozo   +1 more
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Systemic Induced Resistance

2000
Induced resistance is the phenomenon in which a plant, once appropriately stimulated, exhibits an enhanced resistance upon “challenge” inoculation with a pathogen. Induced resistance can be localized as well as systemic, and can be induced by limited pathogen infection, avirulent pathogens, certain non-pathogenic bacteria, and certain chemicals ...
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Nutrient-induced insulin resistance

Molecular and Cellular Endocrinology, 1999
Impaired function of the hormone insulin (insulin resistance) is a major feature of type 2 diabetes, a condition that is expected to afflict over 200 million people by early next century. Intensive investigation has failed to find a genetic basis for insulin resistance in the majority of cases.
J, Proietto   +3 more
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Adaptive response and induced resistance

Comptes Rendus de l'Académie des Sciences - Series III - Sciences de la Vie, 1999
Cellular stress responses are upregulated following exposure to radiation and other DNA-damaging agents. Therefore radiation response can be dose dependent so that small acute exposures (and possibly exposures at very low dose rates?) are more lethal per unit dose than larger exposures above a threshold (typically 10-40 cGy) where induced ...
M C, Joiner, P, Lambin, B, Marples
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The Resistivity and Induced Polarization Methods

Symposium on the Application of Geophysics to Engineering and Environmental Problems 1988, 1988
An attempt is made in this article to provide a readable summary of the bases for and problems encountered with the resistivity and induced polarization methods in gencrat application. The article commences with a discussion of aqueous electrolytic conduction in rocks including consideration of the effects of temperature, rock texture, rock type ...
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Induced Phenotypic Resistance to an Antimetabolite

Science, 1960
Resistance to bacteriostasis by 2-thiazole alanine develops rapidly; however, such resistance is lost during growth in the absence of the analog. This induced resistance is accompanied by increased formation of an enzyme sensitive to 2-thiazole alanine.
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Resistance‐induced antibiotic substitution

Health Economics, 2003
AbstractIn many cases, physicians prescribe antibiotics without knowing whether an individual patient is infected with a susceptible or resistant pathogen. As the proportion of resistant organisms in a community increases, physicians substitute away from older‐inexpensive drugs to newer, more expensive agents as first line therapy.
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