Results 171 to 180 of about 18,601 (330)
Influence of Inhalation Anesthetics on Liver.*
Hans Molitor, Samuel T. Kuna
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Inhalation Anesthetics and Cytochrome P-450-dependent Reactions in Rat Liver Microsomes [PDF]
Bωrje Hallén, Gunnar Johansson
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Trypstatin, a 61‐amino acid Kunitz‐type protease inhibitor derived from human Bikunin, potently inhibits TMPRSS2 and related proteases. It blocks spike‐ and hemagglutinin‐mediated entry of multiple respiratory viruses in vitro reduces SARS‐CoV‐2 replication in human airway cultures, and lowers viral load and symptoms in infected hamsters, demonstrating
Jan Lawrenz+24 more
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Inhalation Anesthetics Augment Oxidant-induced Pulmonary Vasoconstriction [PDF]
Jay R. Shayevitz+4 more
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ABSTRACT Background The pathogenesis of inflammation in eosinophilic chronic rhinosinusitis (ECRS) and non‐eosinophilic chronic rhinosinusitis (NECRS) remains poorly understood. This study aimed to assess immune cell infiltration within the sinonasal microenvironment in these conditions.
Katarzyna Czerwaty+5 more
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INHALATION ANESTHETICS AND SINUS NODE FUNCTION [PDF]
John L. Atlee, T. S. Yeager
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A novel, simple coccygeal intervertebral disc degeneration model, using a suturing method for tail‐looping, without the need for external fixation and complex surgery. The model has high reproducibility and stability and overcomes the disadvantages of the existing animal models of disc degeneration.
Wei Xie+11 more
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Modeling the effect of vorticity on inhaled transport in the upper airway [PDF]
Localized vortices can have significant influence on transport of inhaled particles through the upper respiratory tract. These vortices have complex three-dimensional structure with details dependent on the anatomical geometry. Using a highly simplified model, we demonstrate that changes in transport characteristics with geometric distortion can be ...
arxiv
Establishment of rat model for aspiration pneumonia and potential mechanisms
Aspiration pneumonia was induced in Sprague Dawley rats by intratracheal instillation of hydrochloric acid (HCl, 1.5 μL/g.wt, pH = 1) followed by lipopolysaccharide (LPS, 20 μg/g.wt) after 1 h. The model reproduced hallmark features of acute lung injury, including histological lung damage, microvascular dysfunction, inflammation, hypoxemia, and ...
Hanbing Hu+7 more
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