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Additive effect of multiple genetic variants in <i>SEC23B</i> and <i>PIEZO1</i> on iron metabolism dyshomeostasis in hereditary anemias. [PDF]
HemasphereNostroso A +19 more
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Association between BRINDA-corrected iron metabolism and lung function in the community: the CoLaus|PneumoLaus study. [PDF]
BMC Pulm MedTouilloux B +8 more
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Effects of hydroxy methionine analog iron chelate on growth performance, blood parameters, and iron metabolism in weaned piglets. [PDF]
Sci RepFu Y +9 more
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Current Opinion in Chemical Biology, 1999
The understanding of iron metabolism at the molecular level has been enormously expanded in recent years by new findings about the functioning of transferrin, the transferrin receptor and ferritin. Other recent developments include the discovery of the hemochromatosis gene HFE, identification of previously unknown proteins involved in iron transport ...
P, Aisen +2 more
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The understanding of iron metabolism at the molecular level has been enormously expanded in recent years by new findings about the functioning of transferrin, the transferrin receptor and ferritin. Other recent developments include the discovery of the hemochromatosis gene HFE, identification of previously unknown proteins involved in iron transport ...
P, Aisen +2 more
openaire +3 more sources
Seminars in Pediatric Neurology, 2006
Brain iron uptake is regulated by the expression of transferrin receptor 1 in endothelial cells of the blood-brain barrier. Transferrin-bound iron in the systemic circulation is endocytosed by brain endothelial cells, and elemental iron is released to brain interstitial fluid, likely by the iron exporter, ferroportin.
Tracey A, Rouault, Sharon, Cooperman
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Brain iron uptake is regulated by the expression of transferrin receptor 1 in endothelial cells of the blood-brain barrier. Transferrin-bound iron in the systemic circulation is endocytosed by brain endothelial cells, and elemental iron is released to brain interstitial fluid, likely by the iron exporter, ferroportin.
Tracey A, Rouault, Sharon, Cooperman
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Seminars in Liver Disease, 2005
The liver performs three main functions in iron homeostasis. It is the major site of iron storage, it regulates iron traffic into and around the body through its production of the peptide hepcidin, and it is the site of synthesis of major proteins of iron metabolism such as transferrin and ceruloplasmin.
Gregory J, Anderson, David M, Frazer
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The liver performs three main functions in iron homeostasis. It is the major site of iron storage, it regulates iron traffic into and around the body through its production of the peptide hepcidin, and it is the site of synthesis of major proteins of iron metabolism such as transferrin and ceruloplasmin.
Gregory J, Anderson, David M, Frazer
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La Revue du praticien, 2007
Iron is required for proper cell life functioning. Iron metabolism dysfunction leads in humans to deleterious situations. Maintenance of correct iron plasmatic bioavailability is crucial to permit adequate iron addressing to the different cell types.
Olivier, Loréal +5 more
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Iron is required for proper cell life functioning. Iron metabolism dysfunction leads in humans to deleterious situations. Maintenance of correct iron plasmatic bioavailability is crucial to permit adequate iron addressing to the different cell types.
Olivier, Loréal +5 more
openaire +3 more sources
Epidermal iron metabolism for iron salvage
Journal of Dermatological Science, 2017The epidermis shows a reverse iron gradient from the basal layer to the stratum corneum and consequently, little epidermal intracellular iron is lost by desquamation.To clarify the underlying mechanism of iron salvage.We first used immunohistochemistry and mRNA quantification to demonstrate the distinctive expression pattern of iron metabolism ...
Masayuki, Asano +4 more
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Pediatrics, 1956
THE CLINICIAN has long been aware of the fact that iron deficiency anemia is most prevalent during infancy. Continuing studies of iron metabolism have disclosed many factors which help explain the basic mechanisms of the anemia and the peculiar vulnerability of the infant.
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THE CLINICIAN has long been aware of the fact that iron deficiency anemia is most prevalent during infancy. Continuing studies of iron metabolism have disclosed many factors which help explain the basic mechanisms of the anemia and the peculiar vulnerability of the infant.
openaire +2 more sources