Results 101 to 110 of about 4,311,537 (300)

Modelling of Solar Cell Junction Temperature for Crystalline Silicon Photovoltaic Module

open access: yesSiliconPV Conference Proceedings
Determination of the junction temperature of solar cells is critical for accurately evaluating on-site performance of solar modules. It is difficult to acquire the junction temperature of cells because solar cells are laminated and encapsulated in ...
Zewen Chen   +6 more
doaj  

Numerical simulation of interdigitated back contact hetero-junction solar cells [PDF]

open access: yes, 2013
The goal of this thesis is the application of an opto-electronic numerical simulation to heterojunction silicon solar cells featuring an all back contact architecture (Interdigitated Back Contact Hetero-Junction IBC-HJ).
Callozzo, Alessandro
core  

Regulation of the lncRNA NEAT1 by p53‐ΔNp63 crosstalk modulates the DNA damage response and therapeutic efficacy in HNSCC

open access: yesMolecular Oncology, EarlyView.
In head and neck squamous cell carcinoma (HNSCC) p53 and p63 exert opposite roles on the transcription regulation of the lncRNA NEAT1. Under basal conditions, p53 levels are low and p63 represses NEAT1 expression. Upon genotoxic stress, p53 is rapidly induced, displacing p63 from the NEAT1 promoter leading to NEAT1 transcriptional activation and ...
Sara De Domenico   +5 more
wiley   +1 more source

GEN1 from a thermophilic fungus is functionally closely similar to non-eukaryotic junction-resolving enzymes [PDF]

open access: yes, 2014
Processing of Holliday junctions is essential in recombination. We have identified the gene for the junction-resolving enzyme GEN1 from the thermophilic fungus Chaetomium thermophilum, and expressed the N-terminal 487 amino acid section. The protein is a
Lilley, David M.J.   +8 more
core   +1 more source

Bridging the gap: a genetically validated avian chorioallantoic membrane platform for investigation of spontaneous circulating tumor cells

open access: yesMolecular Oncology, EarlyView.
We established the avian chorioallantoic membrane (CAM) assay as a scalable in vivo model for studying circulating tumor cells (CTCs). Human gastrointestinal tumors spontaneously released genetically validated CTCs that were detected across multiple platforms, demonstrating that the CAM model provides an accessible tool for investigating early cancer ...
Dennis Roth   +17 more
wiley   +1 more source

SPHINX31 acts as a SRPK1 inhibitor targeting the ATR/DNA‐PKcs/CHK1 replicative checkpoint to inhibit cell growth in non‐small cell lung cancer

open access: yesMolecular Oncology, EarlyView.
The kinase SRPK1 directly interacts with the protein TOPBP1 and regulates the pre‐mRNA splicing of WIZ thereby contributing to the activation of the ATR/CHK1 replicative checkpoint in response to replicative stress. This allows cancer cells' genomic stability and survival.
Amani Shreim   +17 more
wiley   +1 more source

Transportation system plan : Junction City, Oregon

open access: yes, 2000
98 pp. Bookmarks modified by UO. Referenced maps not included. Two appendices not included. Published March, 2000. Captured January 4, 2007.The Junction City Transportation System Plan (TSP) is the long-range policy document that guides transportation ...
Lane Council of Governments (Or.)   +1 more
core   +1 more source

Low Temperature Junction Formation for EZ-FET

open access: yesIEEE Journal of the Electron Devices Society
The EZ-FET is a device with simplified architecture and processing that enables fast electrical characterization of semiconductor films on insulators (SOI) with only two lithography levels and using regular process steps.
N. Zerhouni Abdou   +7 more
doaj   +1 more source

Junction Temperature Optical Sensing Techniques for Power Switching Semiconductors: A Review. [PDF]

open access: yesMicromachines (Basel), 2023
Isa R   +4 more
europepmc   +1 more source

Mutant p53R273H disrupts PDPK1 homodimerization and contributes to PDPK1 activation

open access: yesMolecular Oncology, EarlyView.
How mutant p53R273H drives AKT signaling is unclear. We show that p53R273H, but not wild‐type, directly binds PDPK1 via a mutation‐dependent conformational change. This interaction disrupts inhibitory PDPK1 homodimerization and enhances AKT phosphorylation.
Mei Chee Lim   +11 more
wiley   +1 more source

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