Results 151 to 160 of about 15,508 (221)

Subthreshold Cannabidiol Potentiates Levetiracetam in the Kainic Acid Model of Temporal Lobe Epilepsy

Chiara Lucchi, Mattia Marcucci, Kawther Ameen Muhammed Saeed Aledresi, Anna‐Maria Costa, Giuseppe Cannazza, Giuseppe Biagini   +5 more
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The Role of the Neuronal gap Junction Protein Connexin36 in Kainic Acid Induced Hippocampal Excitotoxicity

, 2014
Mark S. Akins
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Cleavage of the TrkB-FL receptor during epileptogenesis: insights from a kainic acid-induced model of epilepsy and human samples.

Pharmacol Res
Ribeiro-Rodrigues L, Fonseca-Gomes J, Paulo SL, Viais R, Ribeiro FF, Miranda-Lourenço C, Mouro FM, Belo RF, Ferreira CB, Tanqueiro SR, Ferreira-Manso M, Umemori J, Castrén E, Paiva VH, Sebastião AM, Aronica E, Campos AR, Bentes C, Xapelli S, Diógenes MJ.   +19 more
europepmc   +1 more source

Strain-Dependent Effects of Sub-chronically Infused Losartan Against Kainic Acid-Induced Seizures, Oxidative Stress, and Heat Shock Protein 72 Expression

, 2013
Jana Tchekalarova, Natasha Ivanova, Daniela Pechlivanova, Kalina Ilieva, Milena Atanasova   +4 more
openalex   +2 more sources

Effects of minocycline on dendrites, dendritic spines, and microglia in immature mouse brains after kainic acid-induced status epilepticus. [PDF]

CNS Neurosci Ther
Yang X, Li T, Liu J, Sun H, Cheng L, Song X, Han Z, Luo H, Han W, Xie L, Jiang L.   +10 more
europepmc   +1 more source

Anti-epileptic and Neuroprotective Effects of Ultra-low Dose NADPH Oxidase Inhibitor Dextromethorphan on Kainic Acid-induced Chronic Temporal Lobe Epilepsy in Rats. [PDF]

Neurosci Bull
Yang JJ, Liu YX, Wang YF, Ge BY, Wang Y, Wang QS, Li S, Zhang JJ, Jin LL, Hong JS, Yin SM, Zhao J.   +11 more
europepmc   +1 more source

Influence of anti-inflammatory interventions in the Kainic acid model of hippocampal excitotoxicity

, 2010
Lorna C. Gleeson
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3H-kainic acid binding: Relevance for evaluating the neurotoxicity of kainic acid

Life Sciences, 1979
Abstract Binding of 3 H-kainic acid (KA) to brain membranes is saturable (K D = 72 nM) and displays markes specificity for compounds that bear relation to excitatory amino acids. Its regional distribution correlates roughly with the regional neurotoxicity of in situ KA injections. Striatal kainate lesions, but not interruption of striatal afferents,
R, Schwarcz, K, Fuxe
openaire   +2 more sources