Results 21 to 30 of about 4,373 (200)

Lambert-Eaton myasthenic syndrome [PDF]

open access: yesCanadian Medical Association Journal, 2006
What's your call? ![Figure][1] Figure. A 72-year-old female smoker presented with morning fatigue, discrete left ptosis and absent reflexes in lower limbs. Top panel: Electromyogram showing evoked motor response of abductor of the fifth finger after stimulation of the left ulnar ...
Monique, D'Amour   +2 more
openaire   +2 more sources

An unusual case of delayed recovery from spinal anesthesia in a patient with Lambert-Eaton myasthenic syndrome—a case report

open access: yesAin Shams Journal of Anesthesiology, 2023
Background The Lambert-Eaton myasthenic syndrome (LEMS) is a rare disorder characterized by proximal muscle weakness and autonomic dysfunction due to involvement of the neuromuscular junction A case of delayed unilateral recovery from spinal anesthesia ...
Souvik Mukherjee   +3 more
doaj   +1 more source

Electrophysiological evaluation of the neuromuscular junction: a brief review

open access: yesArquivos de Neuro-Psiquiatria, 2023
The nerve terminal and muscle membrane compose the neuromuscular junction. After opening the voltage-gated calcium channels, action potentials from the motor axons provoke a cascade for the acetylcholine release from synaptic vesicles to the synaptic ...
João Aris Kouyoumdjian   +1 more
doaj   +1 more source

Lambert-Eaton myasthenic syndrome against the background of thyroid cancer: a clinical case [PDF]

open access: yesКлиническая практика
BACKGROUND: Lambert–Eaton myasthenic syndrome (G73.1 according to ICD-10) is a rare autoimmune disease associated with a presynaptic impairment of neuromuscular transmission due to the production of antibodies to voltage-gated calcium channels.
Elena V. Khozhenko   +5 more
doaj   +1 more source

Lambert–Eaton myasthenic syndrome: Clinical review [PDF]

open access: yes, 2016
Lambert–Eaton myasthenic syndrome (LEMS) is an autoimmune disease of the neuromuscular junction and approximately 60% of LEMS patients have a tumor, mostly small cell lung cancer, as a paraneoplastic neurological syndrome.
Motomura, Masakatsu   +2 more
core   +1 more source

Lambert-Eaton myasthenic syndrome as a cause of persistent neuromuscular weakness after a mediastinoscopic biopsy -A case report- [PDF]

open access: yesKorean Journal of Anesthesiology, 2010
There are many causes of prolonged postoperative muscle weakness, including drugs, residual anesthetics, cerebrovascular events, electrolyte imbalance, hypothermia, and neuromuscular disease.
Cheol Jin Lee   +11 more
doaj   +1 more source

A first case association of Lambert-Eaton Myasthenic Syndrome and First Episode Psychosis: a case report [PDF]

open access: yesEur Psychiatry
Introduction: Lambert-Eaton Myasthenic Syndrome is an autoimmune neuromuscular junction disorder characterized by proximal weakness, autonomic dysfunction, and areflexia associated with antibodies against voltage-gated calcium channels.
Siopa C, Cordeiro C, Moura B.
europepmc   +2 more sources

Successful treatment of advanced lung adenocarcinoma complicated with Lambert‐Eaton myasthenic syndrome: A case report and literature review

open access: yesThoracic Cancer, 2020
Lambert‐Eaton myasthenic syndrome (LEMS) is a rare disease characterized by involvement of the neuromuscular junction. Most cases have an underlying malignancy, especially small‐cell lung cancer (SCLC), while adenocarcinoma is less common.
Aili Wang   +4 more
doaj   +1 more source

Hypotension due to Chemotherapy in a Patient with Small Cell Lung Cancer and Lambert-Eaton Myasthenic Syndrome Undergoing Hemodialysis: A First Case Report

open access: yesCase Reports in Oncology, 2012
We present the first case of small cell lung cancer with Lambert-Eaton myasthenic syndrome during hemodialysis (HD). A 72-year-old male patient receiving HD experienced progressive muscle weakness.
Taiji Kuwata   +3 more
doaj   +1 more source

Lambert‐Eaton Myasthenic Syndrome: A Review [PDF]

open access: yesJournal of General and Family Medicine, 2016
Lambert‐Eaton myasthenic syndrome (LEMS) is an auto‐immune disorder caused by neuromuscular transmission failure, and is a representative pre‐neuromuscular junctional disorder. The auto‐immune antibody is anti‐P/Q‐type voltage‐gated calcium channel (P/Q‐type VGCC) antibody detected in approximately 80 to 90% of LEMS patients.
Matsumoto, Hideyuki, Ugawa, Yoshikazu
openaire   +1 more source

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