Results 51 to 60 of about 6,688 (239)

Review Article : Neuromuscular causes of weakness in critically ill patients [PDF]

open access: yes, 2017
Multiple predisposing factors are responsible for the development of neuromuscular weakness in a critically ill patient. Thorough history and physical examination is paramount and should not be omitted despite that the majority of patients are ...
Govindarajan, Raghav, Katyal, Nakul
core   +1 more source

Pharmacological treatment of Lambert-Eaton Myasthenic Syndrome [PDF]

open access: yes, 2023
Lambert-Eaton myasthenic syndrome (LEMS) is a very rare antibody-mediated autoimmune disease of the neuromuscular junction. Therapy can be divided in symptomatic treatment and immunosuppressive treatment.
Bakker, Wisse Roelof   +5 more
core   +2 more sources

Immune Dysregulation and Self-Reactivity in Schizophrenia: Do Some Cases of Schizophrenia Have an Autoimmune Basis? [PDF]

open access: yes, 2005
Schizophrenia affects 1% of the world's population, but its cause remains obscure. Numerous theories have been proposed regarding the cause of schizophrenia, ranging from developmental or neurodegenerative processes or neurotransmitter abnormalities to ...
Abramsky O   +21 more
core   +1 more source

Lambert-Eaton myasthenic syndrome: the 60th anniversary of Eaton and Lambert's pioneering article

open access: yesArquivos de Neuro-Psiquiatria
This historical review describes the contribution of Drs. Lee M. Eaton and Edward H. Lambert to the diagnosis of myasthenic syndrome on the 60th anniversary of their pioneering article (JAMA 1957) on the disease.
Paulo José Lorenzoni   +3 more
doaj   +1 more source

Paraneoplastic Mononeuritis Multiplex as a Presenting Feature of Adenocarcinoma of the Lung

open access: yesCase Reports in Oncological Medicine, 2013
Paraneoplastic neurologic syndromes are observed in less than 0.1% of cancer patients. Neurologic syndromes in lung cancer include Lambert-Eaton myasthenic syndrome, polyneuropathy, cerebellar degeneration, and rarely mononeuritis multiplex. In this case,
Esra Ekiz   +2 more
doaj   +1 more source

High sensitivity of an ELISA kit for detection of the gamma-isoform of 14-3-3 proteins: usefulness in laboratory diagnosis of human prion disease [PDF]

open access: yes, 2011
Background The gamma-isoform of the 14-3-3 protein (14-3-3 gamma) is expressed in neurons, and could be a specific marker for neuronal damage.
Yuki Matsui   +8 more
core   +1 more source

Centipede venoms as a source of drug leads [PDF]

open access: yes, 2016
peerreview_statement: The publishing and review policy for this title is described in its Aims & Scope. aims_and_scope_url: http://www.tandfonline.com/action/journalInformation?show=aimsScope&journalCode=iedc20© 2016 Taylor and Francis.
de la Vega RC   +6 more
core   +1 more source

Chimeric Antigen Receptor T‐Cells in Myasthenia Gravis: Advances, Safety Challenges, and Future Directions

open access: yesMuscle &Nerve, Volume 73, Issue 6, Page 942-951, June 2026.
ABSTRACT This review examines the emerging application of chimeric antigen receptor (CAR) T‐cell therapy in myasthenia gravis (MG), with emphasis on safety, efficacy signals, and future therapeutic potential in treatment‐refractory disease. A comprehensive literature search was conducted across PubMed, medRxiv, bioRxiv, and Google Scholar for studies ...
Tobias Hegelmaier   +7 more
wiley   +1 more source

Using Intravenous Inmunoglobulin in an Atypical Case of Lambert Eaton Síndrome.

open access: yesMedisur, 2007
A 48 year-old hypertensive, smoker patient who started presenting a generalized weakness, pait instability,visual disorders and uncontrolled blood pressure is presented. At physical examination quadriparesis and weakness extraocular muscles were found. A
Erélido Hernández Valero   +1 more
doaj   +2 more sources

PharmVar GeneFocus: NAT2—Genetic Variation and Updated Nomenclature

open access: yesClinical Pharmacology &Therapeutics, Volume 119, Issue 4, Page 859-871, April 2026.
The Pharmacogene Variation Consortium (PharmVar) provides nomenclature for the highly polymorphic human N‐acetyltransferase 2 (NAT2) gene. NAT2 metabolizes several clinically used drugs including isoniazid, hydralazine, amifampridine, procainamide, and sulfonamides such as dapsone, and also some highly carcinogenic arylamines.
Georgia Papanikolaou   +14 more
wiley   +1 more source

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