Results 291 to 300 of about 96,178 (343)

Phenotype specific nuclear lamina remodeling in hiPSC derived cardiomyocytes bearing <i>TNNT2</i> sarcomeric variants. [PDF]

iScience
Coscarella IL, Usman OH, Wang L, Landim-Vieira M, Wirstiuk L, Brown AN, Zorio DAR, Washburn BK, Vied C, Knollmann BC, Solís C, Galkin VE, Chase PB, Hinson JT, Irianto J, Pinto JR.   +15 more
europepmc   +1 more source

Elevated SUN1 promotes migratory cell polarity defects through mechanically coupling microtubules to the nuclear lamina. [PDF]

Commun Biol
Li Y, Zhang Y, Chen M, Antoku S, Ding J, Huang K, Gundersen GG, Chang W.   +7 more
europepmc   +1 more source

Adipose-derived Stem Cell Sheets Induce Angiogenesis and Hepatic Stellate Cell Activation. [PDF]

In Vivo
Watanabe Y, Kokuryo T, Onoe S, Yamaguchi J, Sunagawa M, Baba T, Kawakatsu S, Watanabe N, Mizuno T, Ebata T.   +9 more
europepmc   +1 more source

The molecular basis of lamin-specific chromatin interactions. [PDF]

Nat Struct Mol Biol
Wang B, Kronenberg-Tenga R, Rosti V, Di Patrizio Soldateschi E, Luo Q, Iannacchero UM, Pinet L, Eibauer M, Boujemaa-Paterski R, Schuler B, Lanzuolo C, Medalia O.   +11 more
europepmc   +1 more source

Manipulation of the nucleoscaffold potentiates cellular reprogramming kinetics. [PDF]

PNAS Nexus
Yang BA, Vesga-Castro C, Monteiro da Rocha A, Newton I, Shcherbina A, Wong SW, Fraczek PM, Larouche JA, Hiraki H, Baker BM, Shin JW, Takayama S, Thouless MD, Aguilar CA.   +13 more
europepmc   +1 more source

Molecular dissection of A-type lamin-regulated pathways

, 2017
Tobias Corne
openalex   +1 more source

The accumulation of progerin underlies the loss of aortic smooth muscle cells in Hutchinson-Gilford progeria syndrome. [PDF]

Cell Death Dis
Kim PH, Kim JR, Heizer PJ, Jung H, Tu Y, Presnell A, Scheithauer J, Yu RG, Young SG, Fong LG.   +9 more
europepmc   +1 more source

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Invertebrate lamins

Experimental Cell Research, 2007
Lamins are the main component of the nuclear lamina and considered to be the ancestors of all intermediate filament proteins. They are localized mainly at the nuclear periphery where they form protein complexes with integral proteins of the nuclear inner membrane, transcriptional regulators, histones and chromatin modifiers.
Shai, Melcer, Yosef, Gruenbaum, Georg, Krohne   +2 more
openaire   +2 more sources