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Lentivirus infection of macrophages.
Immunology series, 1994The ovine and caprine lentiviruses infect monocytes, and the viral DNA is integrated into the cellular DNA. The provirus remains silent until the monocyte matures into a macrophage. Intrinsic to this maturation is the induction of a class of immediate early genes in the monocyte that includes the transcription factors JUN and FOS.
J E, Clements +3 more
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Unique lentivirus-host interactions: SIVsmmPBj14 infection of macaques
Virus Research, 1994The most virulent primate lentivirus identified to date, the simian virus SIVsmmPBj14 (SIV-PBj14), is unique not only because it causes acute disease and death within days instead of months or years, but also because of its replicative and cellular activation properties. The acute disease syndrome has many features in common with primary HIV-1 disease,
P N, Fultz, P M, Zack
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NEF: A Pleiotropic Modulator of Primate Lentivirus Infectivity and Pathogenesis
Acta Microbiologica et Immunologica Hungarica, 2006The lentiviral protein Nef recruits cellular signalling proteins to lipid rafts at the cell membrane and acts thereby as a master regulator affecting the transcription of a series of cellular genes. By activating resting T cells, Nef creates an optimal environment for lentivirus replication.
Balog, Katalin, Minarovits, J.
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In vivo activation of alveolar macrophages in ovine lentivirus infection
Clinical Immunology and Immunopathology, 1990Sheep infected by visna-maedi virus, a lentivirus related to the human immunodeficiency virus, develop a chronic interstitial lung disease. Since monocyte/macrophages are known to be specifically infected by visna-maedi virus, we investigated the role of macrophages in the appearance of pulmonary lesions in animals with naturally occurring disease ...
Cordier, Geneviève +7 more
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Immunoblot analysis of the antibody response to ovine lentivirus infections
Veterinary Microbiology, 1989The antibody response of sheep to maedi-visna virus (MVV) infection was studied, using an immunoblotting technique that identified the four major viral structural proteins: the envelope glycoprotein gp135, and internal proteins p25, p16 and p14. In sequential serum samples of two inoculated sheep, antibodies to p25 appeared first, soon followed by ...
D J, Houwers, I M, Nauta
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[Pathological findings in lentivirus infected cats].
Schweizer Archiv fur Tierheilkunde, 1989This report describes the necropsy findings in 3 cats with a natural T-lymphotropic Lentivirus (FTLV) infection. The changes are compared with the "immunodeficiency syndrome" of humans and monkeys. Furthermore, the pathogenicity of the FTLV and the significance of a dual infection with FTLV and Spumavirus is discussed.
V, Dieth, H, Lutz, B, Hauser, P, Ossent
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Early events in infection of lymphoid tissue by a lentivirus, maedi-visna
Trends in Microbiology, 1995All members of the lentivirus family infect cells of the monocyte-macrophage lineage, although this may be obscured during infection in vivo by the effect of the virus on other cells, such as CD4+ lymphocytes. Macrophages are the major cell type infected by the ruminant lentivirus maedi-visna, making it a valuable model for studying the pathogenesis of
B, Blacklaws, P, Bird, I, McConnell
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[Infections with feline T-lymphotropic lentivirus].
Tijdschrift voor diergeneeskunde, 1988A new retrovirus was recently discovered in cats affected with an immunodeficiency syndrome (AIDS). The virus shows morphological and biological similarities with the human immunodeficiency virus (HIV), which causes AIDS in human individuals. As the T-lymphocyte is the primary target of the virus, it is termed Feline T-Lymphotropic Lentivirus (FTLV ...
H, Egberink +4 more
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Feline lentivirus infection in nondomestic felids.
Seminars in veterinary medicine and surgery (small animal), 1996Human immunodeficiency virus and feline immunodeficiency virus are both lentiviruses that cause immunosuppressive disease. Similarities between these diseases have promoted the study of feline immunodeficiency virus as a model for human immunodeficiency virus.
A N, Plotnick, R S, Larsen
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Lentivirus infection of macrophages
2018Several aspects of the interaction of macrophages and maedi visna virus (MVV) were undertaken: viral replication, phenotype, phagocytosis and antigen presenting function of macrophages after MVV (EV1) infection. MVV replication in monocyte-derived-macrophages (MDM) showed viral budding sites both at cytoplasmic and vesicular membranes.
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