Results 211 to 220 of about 76,259 (245)

Predisposition to hematopoietic malignancies by deleterious germline CHEK2 variants. [PDF]

Leukemia
Stubbins RJ, Arnovitz S, Vagher J, Asom A, Perpich M, Pies M, Akpan IJ, Chew E, Bridgers J, Karsan A, Rodgers C, Koppayi A, Basdag H, Drazer MW, Das S, Cheng J, Osman AEG, Godley LA.   +17 more
europepmc   +1 more source

Spectrum of Cytogenetic Abnormalities in Adolescent and Young Adult Patients of B-Cell Acute Lymphoblastic Leukemia in a Tertiary Care Hospital in Pakistan. [PDF]

Cureus
Brohi S, Shaikh IA, Tariq F, Tahir A, Iftikhar I, Siddiqui IA, Bokhari SW, Tariq Mahmood M, Qazi R, Ahmad U.   +9 more
europepmc   +1 more source

The protein deacetylase HDAC10 controls DNA replication in malignant lymphoid cells. [PDF]

Leukemia
Mieland AO, Petrosino G, Dejung M, Chen JX, Fulzele A, Mahmoudi F, Tu JW, Mustafa AM, Zeyn Y, Hieber C, Bros M, Schnöder TM, Heidel FH, Najafi S, Oehme I, Hofmann I, Schutkowski M, Hilscher S, Kosan C, Butter F, Bhatia S, Sippl W, Krämer OH.   +22 more
europepmc   +1 more source

Clinicohematological Profile of Pancytopenia Patients in Tertiary Care Hospital.

J Pharm Bioallied Sci
Wadhera D, Naryal P, Mishra T, Singh J, Bali IK.   +4 more
europepmc   +1 more source

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Acute lymphoid leukemia etiopathogenesis

Molecular Biology Reports, 2021
Acute lymphoid leukemia (ALL) is a type of hematological neoplasm that affects the precursor cells of strains B, T  and NK, with a higher incidence in the pediatric range. The pathophysiology of ALL is characterized by chromosomal abnormalities and genetic alterations involved in the differentiation and proliferation of lymphoid precursor cells ...
Thiago Cezar Fujita, Nathália Sousa-Pereira, Marla Karine Amarante, Maria Angelica Ehara Watanabe   +3 more
openaire   +3 more sources

Acute Non-Lymphoid Leukemia

Pediatric Annals, 1983
Acute non-lymphoid leukemia is a group of hematologic neoplasms which have been the subject of intensive basic and clinical research. These studies have led to a better understanding of the genetic basis of leukemia and may ultimately help establish the molecular mechanisms of malignant transformation.
Robert R. Chilcote, Carlton Dampier
openaire   +3 more sources

Lonidamine in the Treatment of Chronic Lymphoid Leukemia

Oncology, 1984
Blood and/or bone marrow cells from patients with hematological malignancies, chronic lymphocytic leukemia (CLL), chronic myeloid leukemia in blastic phase, acute lymphoid leukemia, acute myeloid leukemia, prolymphocytic leukemia, multiple myeloma and myelofibrosis, were incubated with Lonidamine at different concentrations (50, 100 and 160 micrograms ...
Tura S, Cavo M, GOBBI, MARCO, Franchi P.
openaire   +4 more sources

Acute Lymphoid Leukemia

Pediatric Annals, 1983
During the past decade progress in the treatment of childhood acute lymphoblastic leukemia has slowed. A 50% to 60% cure barrier has frustrated a multitudinous array of therapeutic attempts to overcome this obstacle. With few exceptions, intensifications of induction, consolidation, or maintenance therapies have not overcome this obstacle.
openaire   +2 more sources

Chronic lymphoid leukemia

Hématologie, 2013
hma.2013.0854 Auteur(s) : Bruno Cazin b-cazin@chru-lille.fr Service des maladies du sang, CHRU de Lille, Hopital Huriez, Lille Comment traiter une leucemie lymphoide chronique (LLC) ? Avant de repondre a cette question, se pose une question prealable : dois-je traiter ce patient atteint de LLC ?
openaire   +2 more sources