Results 131 to 140 of about 17,237 (190)

Flavoprotein Fluorescence Imaging in Stargardt Disease: Linking Metabolic Stress to Structural Damage. [PDF]

Invest Ophthalmol Vis Sci
Merle DA, Cuevas Villanueva V, Righetti G, Jung R, Kempf M, Kohl S, Wissinger B, Kühlewein L, Stingl K, Stingl K.   +9 more
europepmc   +1 more source

Emerging Radioligands as Tools to Track Multi-Organ Senescence. [PDF]

Diagnostics (Basel)
Gagliardi A, Migliari S, Guercio A, Baldari G, Graziani T, Cervati V, Ruffini L, Scarlattei M.   +7 more
europepmc   +1 more source

Identification of active anti-aging ingredients from asparagus cochinchinensis based on the spectrum-effect relationship. [PDF]

BMC Complement Med Ther
Wan R, Cai Z, Liu Z, You J, Wang L, Tang X, Chen Y, Wang Q, Ou L, Zhang C.   +9 more
europepmc   +1 more source

Corneal stromal microdots accumulation and its association with corneal neurodegeneration and retinal microvascular perfusion in diabetes. [PDF]

Front Endocrinol (Lausanne)
Wang J, Wang S, Zhang H, Jin X.
europepmc   +1 more source

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Lipofuscin

International Journal of Biochemistry and Cell Biology, 2004
Over time, postmitotic cells accumulate a non-degradable intralysosomal substance, lipofuscin, which forms due to iron-catalyzed oxidation/polymerization of protein and lipid residues. Lipofuscin is often considered a hallmark of aging, showing an accumulation rate that inversely correlates with longevity.
Alexei, Terman, Ulf T, Brunk
exaly   +4 more sources

Lipofuscin

Annals of the New York Academy of Sciences, 2007
Abstract:  One of the highlights of postmitotic aging is the intracellular accumulation of highly oxidized and cross‐linked proteins, known as lipofuscin. Lipofuscin is insoluble and not degradable by lysosomal enzymes or the proteasomal system, which is responsible for the recognition and degradation of misfolded and oxidatively damaged proteins ...
Tobias Jung, Tilman Grune
exaly   +3 more sources