Results 21 to 30 of about 309,429 (287)

A comparison of parameter choice rules for lp - lq minimization [PDF]

open access: yes, 2022
Images that have been contaminated by various kinds of blur and noise can be restored by the minimization of an lp-lq functional. The quality of the reconstruction depends on the choice of a regularization parameter.
Sgallari F.   +3 more
core   +1 more source

A quantitative assay for the non-covalent association between apolipoprotein[a] and apolipoprotein B: an alternative measure of Lp[a] assembly

open access: yesJournal of Lipid Research, 2000
Increasing evidence suggests that the assembly of lipoprotein[a] (Lp[a]) proceeds in two steps. In the first step, non-covalent interactions between apolipoprotein[a] (apo[a]) and apolipoprotein B (apoB) of low density lipoprotein (LDL) form a ...
B.N. Dardik   +3 more
doaj   +1 more source

Lipoprotein(a) accelerates atherosclerosis in uremic mice[S]

open access: yesJournal of Lipid Research, 2010
Uremic patients have increased plasma lipoprotein(a) [Lp(a)] levels and elevated risk of cardiovascular disease. Lp(a) is a subfraction of LDL, where apolipoprotein(a) [apo(a)] is disulfide bound to apolipoprotein B-100 (apoB).
Tanja X. Pedersen   +4 more
doaj   +1 more source

LPS Algorithms

open access: yes, 1984
LPS is a Logic Programming System currently under development and specially targeted for implementation on massively parallel architectures. We present a detailed explanation of algorithms under development for parallel execution of LPS programs. The explanation is significantly more detailed then those published previously. An abstract proof procedure
Lowry, Andy   +2 more
openaire   +2 more sources

Lp(a):a New Pathway to Target?

open access: yesCurrent atherosclerosis reports, 2022
Purpose of Review: Over the past decades, genetic and observational evidence has positioned lipoprotein(a) as novel important and independent risk factor for cardiovascular disease (ASCVD) and aortic valve stenosis. Recent Findings: As Lp(a) levels are determined genetically, lifestyle interventions have no effect on Lp(a)-mediated ASCVD risk.
Nurmohamed, Nick S.   +2 more
openaire   +2 more sources

Realizations and LP

open access: yesAnnals of Pure and Applied Logic, 2007
Abstract LP  can be seen as a logic of knowledge with justifications. See [S. Artemov, The logic of justification, The Review of Symbolic Logic 1 (4) (2008) 477–513] for a recent comprehensive survey of justification logics generally. Artemov’s Realization Theorem says justifications can be extracted from validities in the more conventional Hintikka-
openaire   +1 more source

Lp-PLA 2 , a new biomarker of vascular disorders in metabolic diseases [PDF]

open access: yes, 2019
Metabolic diseases are chronic disorders correlated to a greater risk of cardiovascular event and death. Recently, many data have sustained the biological link between microvascular dysfunction, oxidative stress, vascular inflammation, and metabolic ...
Rovella V   +19 more
core   +1 more source

Diet and Lp(a): Does Dietary Change Modify Residual Cardiovascular Risk Conferred by Lp(a)? [PDF]

open access: yes, 2020
Lipoprotein(a) [Lp(a)] is an independent, causal, genetically determined risk factor for cardiovascular disease (CVD). We provide an overview of current knowledge on Lp(a) and CVD risk, and the effect of pharmacological agents on Lp(a). Since evidence is
Enkhmaa, Byambaa,; Petersen, Kristina S,; Kris-Etherton, Penny M,; Berglund, Lars,   +8 more
core   +1 more source

Role of Lp(a) in patients with erectile dysfunction undergoing angioplasty for symptomatic pelvic artery disease.

open access: yes, 2023
Background: Atherosclerotic disease of erection-related arteries is a major reason for erectile dysfunction (ED). Lp(a) has been implied in the pathophysiology of atherosclerosis in the coronary and lower limb arteries.
Hak-Hong Keo   +13 more
core   +1 more source

High lipoprotein(a): Actionable strategies for risk assessment and mitigation

open access: yesAmerican Journal of Preventive Cardiology
High levels of lipoprotein(a) [Lp(a)] are causal for atherosclerotic cardiovascular disease (ASCVD). Lp(a) is the most prevalent inherited dyslipidemia and strongest genetic ASCVD risk factor.
Gissette Reyes-Soffer   +4 more
doaj   +1 more source

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