Results 171 to 180 of about 13,010 (203)

Perturbation of BRD4 and p300 activity suppresses super enhancer‑driven <i>KLF6</i> expression in renal carcinoma. [PDF]

Oncol Rep
Zamberi NNM, Abuhamad AY, Yusuf SNHM, Rahman NQAA, Mohtar MA, Syafruddin SE.   +5 more
europepmc   +1 more source

FUS is an N1- and N6-methyladenosine-binding protein. [PDF]

Nucleic Acids Res
Liang X, Zhao T, Dai X, Sun Y, Yuan J, Afzalpurkar S, Duong C, Yu A, Tang F, He X, Liu X, Chen X, Cao Z, Wang Y.   +13 more
europepmc   +1 more source

Association of primary lesion surgery and surgical modality with overall survival in patients with stage IV cutaneous melanoma: a population-based study. [PDF]

Transl Cancer Res
Mo DC, Chen L, Du YJ, Luo PH, Huang SX, Wang HL, Liang XJ, Lin P, Huang JF.   +8 more
europepmc   +1 more source

Some of the next articles are maybe not open access.

Related searches:

Dysregulation of the Epitranscriptomic Mark m1A in Ischemic Stroke

Translational Stroke Research, 2022
Methylation of adenosine at N1 position yields N1-methyladenosine (m1A), which is an epitranscriptomic modification that regulates mRNA metabolism. Recent studies showed that altered m1A methylation promotes acute and chronic neurological diseases. We currently evaluated the effect of focal ischemia on cerebral m1A methylome and its machinery.
Anil K Chokkalla, Raghu Vemuganti, Qing Dai   +2 more
exaly   +3 more sources

The Impact of m1A Methylation Modification Patterns on Tumor Immune Microenvironment and Prognosis in Oral Squamous Cell Carcinoma

International Journal of Molecular Sciences, 2021
N1-methyladenosine (m1A) modification widely participates in the occurrence and progression of numerous diseases. Nevertheless, the potential roles of m1A in the tumor immune microenvironment (TIME) are still not fully understood.
Yo Kishimoto, Masahiro Sugimoto, Koichi Omori   +2 more
exaly   +2 more sources

Mo’ m1A, mo’ problems

Cell Chemical Biology
CAG-repeat expansions underlie fatal neurodegenerative disorders. In a lodestar study published in a recent issue of Nature, Sun et al.1 identify a writer and eraser of N1-methyladenosine (m1A) modifications of CAG-repeat RNA. They establish that m1A modifications in CAG-repeat expanded RNA promote neurodegeneration and aberrant phase transitions of ...
Linamarie, Miller, James, Shorter
openaire   +2 more sources

M1A - M.I.A.? [PDF]

, 1987
An examination of the compositional changes in the monetary aggregates M1 and M1A, with a discussion of their behavior in the 1980s and some issues relevant to the possibility of replacing M1 with M1A in the monetary targeting framework.
William T. Gavin, Michael R. Pakko
openaire  

Mapping m6A and m1A with mutation signatures

Nature Methods, 2019
In two novel RNA modification mapping methods, the authors have engineered RNA enzymes and used the enzyme-mediated mutational signatures to map m6A and m1A at single-nucleotide resolution in mammalian RNA.
openaire   +2 more sources

Monetarism Beyond M1A [PDF]

, 2004
This work analyzes, from an empirical standpoint, the information contained in the M1A aggregate in Chile and compares it with other aggregates. The findings reveal that, at least for now, the M1A aggregate does not seem to have particularly valuable information that would make it a better indicator of inflationary pressures within the monetary policy ...
Pablo García,, Rodrigo O. Valdés
openaire  

The m1A landscape on cytosolic and mitochondrial mRNA at single-base resolution

Nature, 2017
Modifications on mRNA offer the potential of regulating mRNA fate post-transcriptionally. Recent studies suggested the widespread presence of N1-methyladenosine (m1A), which disrupts Watson-Crick base pairing, at internal sites of mRNAs. These studies lacked the resolution of identifying individual modified bases, and did not identify specific sequence
Modi Safra, Aldema Sas-Chen, Ronit Nir, Roni Winkler, Aharon Nachshon, Dan Bar-Yaacov, Matthias Erlacher, Walter Rossmanith, Noam Stern-Ginossar, Schraga Schwartz   +9 more
openaire   +3 more sources