Results 221 to 230 of about 994,457 (252)
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Melanocortin 1 receptor variants, pigmentation, and skin cancer susceptibility

Photodermatology, Photoimmunology & Photomedicine, 2004
The melanocortin 1 receptor is a key regulator of variation in normal human pigmentation. Genetic variants of this receptor cause red hair and fair skin, and several case–control studies have demonstrated that these genetic variants increase the risk of skin cancer development in humans.
openaire   +2 more sources

[Genotype, melanocortin 1 receptor and ultraviolet radiation].

Yi chuan = Hereditas, 2005
Recent work on the melanocortin 1 receptor (MC1R) suggests that MC1R plays a central role in regulation of eumelanin (brown/black melanins) and phaeomelanin (red/yellow melanins) synthesis within the mammalian melanocyte. In the mouse,genetic studies show that the MC1R appears to be a unique, bifunctionally controlled receptor, activated by alpha-MSH ...
Xue-mei, Lü, Peng, Shi, Ya-ping, Zhang
openaire   +1 more source

Critical care management of chimeric antigen receptor T‐cell therapy recipients

Ca-A Cancer Journal for Clinicians, 2022
Alexander Shimabukuro-Vornhagen   +2 more
exaly  

MC-1 Melanocortin Receptor

2007
S.J. Enna, David B. Bylund
openaire   +1 more source

Targeting melanocortin receptor type 1 with small peptides

British Journal of Dermatology, 2019
A.T. Slominski   +2 more
openaire   +2 more sources

Melanocortin 1 receptor (MC1R) polymorphisms’ influence on size and dermoscopic features of nevi

Pigment Cell & Melanoma Research, 2018
M. G. Vallone   +9 more
semanticscholar   +1 more source

Preclinical evaluation of melanocortin‐1 receptor (MC1‐R) specific 68Ga‐ and 44Sc‐labeled DOTA‐NAPamide in melanoma imaging

European Journal of Pharmaceutical Sciences, 2017
G. Nagy   +9 more
semanticscholar   +1 more source

Assessment of gene expression levels of proopiomelanocortin (POMC) and melanocortin‐1 receptor (MC1R) in vitiligo

Australasian Journal of Dermatology, 2017
N. Nagui   +4 more
semanticscholar   +1 more source

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