Results 231 to 240 of about 248,446 (274)

Polygenic Mendelian Randomization

Cold Spring Harbor Perspectives in Medicine, 2020
Many exposures considered in Mendelian randomization (MR) studies are polygenic in that they are influenced by thousands of genetic variants. By using many single-nucleotide polymorphisms (SNPs) as instrumental variables, more variation in the exposure is explained, increasing the precision of MR.
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Mendelian Inheritance

Postgraduate Medicine, 1972
The first approach to analysis of the inheritance pattern of mendelian diseases depends on whether the mutant gene or genes are located on one of the 44 (22 pairs) autosomes or on the one or two X chromosomes. No human disease is known to be associated with a mutant gene on the Y chromosome.
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Mendelian and non-Mendelian heredity: a reappraisal

Proceedings of the Royal Society of London. Series B. Biological Sciences, 1966
The first report of a non-Mendelian gene (Correns 1908) appeared just eight years after the rediscovery of Mendel’s laws and, in this sense, Mendelian and non-Mendelian heredity have both been available for analysis and comparison for almost sixty years.
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MENDELIAN HYPERPHENYLALANINEMIA

Annual Review of Genetics, 1988
C R, Scriver, S, Kaufman, S L, Woo
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Mendelian inheritance

Nature Ecology & Evolution, 2022
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Mendelian Randomization Concerns

JAMA Psychiatry, 2018
Lin, Bochao Danae, Li, Yue, Luykx, Jurjen   +2 more
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Mendelian Differences

Science, 1931
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Mendelian Heredity

Science, 1908
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Plasma HDL cholesterol and risk of myocardial infarction: a mendelian randomisation study

Lancet, The, 2012
Benjamin F Voight, Ruth Frikke-Schmidt, Maja Barbalic   +2 more
exaly  

Mendelian, non-Mendelian, multigenic inheritance, and epigenetics

2020
Tamar Harel, James R. Lupski
openaire   +1 more source