Results 321 to 330 of about 400,251 (340)

Role of AMP-Activated Protein Kinase (AMPK) in Female Reproduction: A Review. [PDF]

open access: yesInt J Mol Sci
Kamar Bashah NA   +5 more
europepmc   +1 more source

mTOR Modulates NLRP3 Inflammasome Activation via Nuclear Translocation and STAT1 Inhibition. [PDF]

open access: yesEur J Immunol
González-Dominguez A   +6 more
europepmc   +1 more source

Treinamento de força versus treinamento de endurance. Existe compatibilidade?

open access: yesRevista Brasileira de Prescrição e Fisiologia do Exercício, 2012
Bernardo Neme Ide   +7 more
doaj  

mTOR Pathway and mTOR Inhibitors in Cancer Therapy [PDF]

open access: possible, 2010
1. N. Kubica and J. Blenis "mTORC1: A signaling integration node involved in cell growth" 2. Z. Feng and A.J. Levine "The Regulation of the IGF-1/mTOR Pathway by the p53 Tumor Suppressor Gene Functions" 3. H. Mead, M. Zeremski and M. Guba "mTOR Signaling in Angiogenesis" 4. J. Brugaroles "mTOR signaling and hypoxia" 5. D. Akhavan and P.S. Mischel "mTOR
Vitaly A. Polunovsky, Peter J. Houghton
openaire   +1 more source

mTOR Signaling Pathway and mTOR Inhibitors in Cancer Therapy

Hematology/Oncology Clinics of North America, 2012
Mammalian target of rapamycin (mTOR) is a serine/threonine protein kinase. It is ubiquitously expressed in cells and is a therapeutic target for the cancer treatment arsenal. Despite the great responses obtained in tumors addicted to specific mutations or overactivation of key members of the mTOR pathway (HiF1α in RCC, cyclin D1 in MCL, or TSC in SEGA),
Alejandro Gomez-Pinillos   +1 more
openaire   +3 more sources

mTOR Pathway and mTOR Inhibitors as Agents for Cancer Therapy

Current Cancer Drug Targets, 2008
Research into mTOR, mammalian Target Of Rapamycin as an important drug target continues to be extremely interesting, both in terms of the increased molecular knowledge being acquired at the basis of various human diseases, and also for possible applications in drug cancer therapy.
Renzo Lazzarini   +5 more
openaire   +3 more sources

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