Results 311 to 320 of about 65,558 (345)
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Chlordane as a Neurotoxin in Humans
Southern Medical Journal, 1997To assay and profile chronic neurobehavioral impairment associated with chlordane exposure in symptomatic patients, consecutive evaluations of nine patients were done with sensitive neurophysiologic and neuropsychologic tests for neurobehavioral function. Their visual fields, balance, reaction time, blink, color discrimination, grip strength, cognitive
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Neurotoxins and Neurodevelopment
2016The leading cause of neonatal mortality is birth defects, in some cases the result of exposure to neurotoxins. Even when not resulting in death, damage to the developing brain or central nervous system as a result of exposure can result in negative impacts that affect lifelong outcomes.
Morgan B. Drake +2 more
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Acta Psychiatrica Scandinavica, 1983
The role of toxic substances in causing central nervous system dysfunction is discussed with a focus on effects which are mediated indirectly through other organ systems which affect the brain. Neurochemical measurements of brain function, and the use of neuropharmacological probes of behavior are presented with the examples of lead poisoning, as a ...
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The role of toxic substances in causing central nervous system dysfunction is discussed with a focus on effects which are mediated indirectly through other organ systems which affect the brain. Neurochemical measurements of brain function, and the use of neuropharmacological probes of behavior are presented with the examples of lead poisoning, as a ...
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Monoaminergic neurotoxins: Are they selective?
Journal of Neural Transmission, 1975Introduction of 6-hydroxydopamine into the cerebrospinal fluid produces a selective destruction of adrenergic pathways. However, the selectivity of interstitial injections of 6-hydroxydopamine for catecholamine elements is critically dependent upon the dose and volume of injection must be evaluated by biochemical and cytochemical indices.
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6-Hydroxydopamine: a far from simple neurotoxin
Journal of neural transmission, 2020D. Varešlija +3 more
semanticscholar +1 more source
Neurologic Clinics, 2000
The epidemiology, clinical features, pathology, and mechanisms of action of basal ganglia neurotoxins are reviewed. Manganese, cyanide, hydrogen sulfide, methanol, carbon monoxide, 3-nitropropionic acid, MPTP, and annonaceae alkaloids are discussed. The probable mechanism of action for almost all basal ganglia neurotoxins is inhibition of mitochondrial
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The epidemiology, clinical features, pathology, and mechanisms of action of basal ganglia neurotoxins are reviewed. Manganese, cyanide, hydrogen sulfide, methanol, carbon monoxide, 3-nitropropionic acid, MPTP, and annonaceae alkaloids are discussed. The probable mechanism of action for almost all basal ganglia neurotoxins is inhibition of mitochondrial
openaire +3 more sources