Results 341 to 350 of about 345,929 (388)
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Modelling oxide–oxide and oxide–metal interfaces
J. Chem. Soc., Faraday Trans., 1990Recent years have seen a large increase in interest in interfaces of ionic and ceramic crystals and an appreciation of their importance in determining many of the properties of materials. Static-lattice calculations have made a major impact on the interpretation of the role of point defects in controlling observed behaviour in the bulk and are now ...
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Annals of the New York Academy of Sciences, 2000
Abstract: The oxidative modification of proteins by reactive species, especially reactive oxygen species, is implicated in the etiology or progression of a panoply of disorders and diseases. These reactive species form through a large number of physiological and non‐physiological reactions.
E R, Stadtman, R L, Levine
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Abstract: The oxidative modification of proteins by reactive species, especially reactive oxygen species, is implicated in the etiology or progression of a panoply of disorders and diseases. These reactive species form through a large number of physiological and non‐physiological reactions.
E R, Stadtman, R L, Levine
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Baillière's Clinical Endocrinology and Metabolism, 1995
The understanding of the role of lipoprotein oxidation is still incomplete. Much is still to be learned about the mechanism of action of oxidized lipoproteins on different types of cell, as well as the origin of the oxidation process, and how it links to the situation in vivo.
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The understanding of the role of lipoprotein oxidation is still incomplete. Much is still to be learned about the mechanism of action of oxidized lipoproteins on different types of cell, as well as the origin of the oxidation process, and how it links to the situation in vivo.
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Annals of the New York Academy of Sciences, 2000
Abstract: There is a large body of evidence showing both metabolic defects and oxidative damage in Alzheimer's disease. Studies of cybrid cell lines show reduced cytochrome oxidase. There is also substantial evidence for a defect in a‐ketoglutarate dehydrogenase.
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Abstract: There is a large body of evidence showing both metabolic defects and oxidative damage in Alzheimer's disease. Studies of cybrid cell lines show reduced cytochrome oxidase. There is also substantial evidence for a defect in a‐ketoglutarate dehydrogenase.
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2010
The immediate source of energy for muscle contraction is provided by the hydrolysis of adenosine triphosphate (ATP). However, muscle ATP stores are not large and would be consumed in only a few seconds of high-intensity exercise if they were not quickly replenished.
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The immediate source of energy for muscle contraction is provided by the hydrolysis of adenosine triphosphate (ATP). However, muscle ATP stores are not large and would be consumed in only a few seconds of high-intensity exercise if they were not quickly replenished.
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Radical generation by the interaction of transition metals with common oxidants.
Environmental Science and Technology, 2004George P. Anipsitakis, D. Dionysiou
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Oxidants, oxidative stress and the biology of ageing
Nature, 2000Toren Finkel, Nikki J. Holbrook
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Current Protocols in Cytometry, 1997
AbstractPeroxide production is detected using DCFH‐DA, superoxide anion by hydroethidine; the methods can be combined for simultaneous detection. A support protocol describes calibration of the flow cytometer to permit calculation of the actual H2O2 production per cell.
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AbstractPeroxide production is detected using DCFH‐DA, superoxide anion by hydroethidine; the methods can be combined for simultaneous detection. A support protocol describes calibration of the flow cytometer to permit calculation of the actual H2O2 production per cell.
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Oxidation and oxidation protection
1993Over the past decade the development of structural carbon-carbon materials has received a great deal of attention. Potential uses have been cited in future generation military aircraft, missile systems and a number of proposed hypersonic aerospace vehicles.
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Oxidants, oxidant drugs, and malaria
Journal of Medicinal Chemistry, 1988J L, Vennerstrom, J W, Eaton
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