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Correction to “Monoamine Oxidase‐A Is a Novel Driver of Stress‐Induced Premature Senescence Through Inhibition of Parkin‐Mediated Mitophagy”

Aging Cell, Volume 25, Issue 5, May 2026.
wiley   +1 more source

Influence of Parkin Specific Domain on Parkin Ubiquitination

, 2018
openaire   +1 more source

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No Parkin Zone: Mitophagy without Parkin

Trends in Cell Biology, 2018
Mitochondria are essential highly dynamic organelles that provide the necessary energy for a variety of different processes, such as survival, proliferation, and migration. In order to maintain an intact mitochondrial network, cells have developed quality control systems that allow the removal of damaged or superfluous mitochondria by selective ...
Elodie, Villa, Sandrine, Marchetti, Jean-Ehrland, Ricci   +2 more
openaire   +2 more sources

Parkin and mitochondrial signalling

Cellular Signalling, 2023
Aging, toxic chemicals and changes to the cellular environment are sources of oxidative damage to mitochondria which contribute to neurodegenerative conditions including Parkinson's disease. To counteract this, cells have developed signalling mechanisms to identify and remove select proteins and unhealthy mitochondria to maintain homeostasis.
Elizabeth M, Connelly, Karling S, Frankel, Gary S, Shaw   +2 more
openaire   +2 more sources

Parkin gene therapy

Parkinsonism & Related Disorders, 2009
The search for genetic mutations responsible for familial forms of Parkinson's disease (PD) has identified several genes and loci. Mutations in the parkin gene are linked to autosomal recessive juvenile parkinsonism. The genetic forms of PD are uncommon, but gene therapy targeting alpha-synuclein, parkin, or other pathways may be also applicable for ...
openaire   +2 more sources