Results 251 to 260 of about 165,634 (303)
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Pemphigus Vulgaris

Diseases of the Oral Mucosa, 2021
M. Kasperkiewicz, Enno Schmidt
semanticscholar   +2 more sources

The involvement of ADAM10 in acantholysis in mucocutaneous pemphigus vulgaris depends on the autoantibody profile of each patient

British Journal of Dermatology, 2020
Acantholysis in pemphigus vulgaris (PV) may be triggered by desmoglein (Dsg) and non‐Dsg autoantibodies. The autoantibody profile of each patient results in distinct intracellular signalling patterns.
M. Ivars   +5 more
semanticscholar   +1 more source

Pemphigus Vulgaris

The Journal of the American Dental Association, 1980
Pemphigus vulgaris is a rare, potentially fatal skin disease with lesions usually first appearing in the mouth. Histologic examination is the only reliable mechanism with which to establish an accurate diagnosis. An elderly patient with multiple oral and skin lesions was diagnosed, referred to the dermatology service, and successfully treated with ...
P L, Kempler, T R, Schott
openaire   +2 more sources

Pemphigus Vulgaris and Paraneoplastic Pemphigus

Oral and Maxillofacial Surgery Clinics of North America, 2008
The history, epidemiology, pathophysiology, clinical presentation, diagnostic work-up, histopathology, and treatment of PV and PNP have been presented. These life-threatening, autoimmune, mucocutaneous bullous conditions may be encountered first by oral health providers and, therefore, deserve keen understanding and attention by the oral and ...
Wendell A, Edgin   +2 more
openaire   +2 more sources

Juvenile Pemphigus Vulgaris

Acta Paediatrica, 1991
ABSTRACT. Pemphigus vulgaris is extremely rare in childhood. Only 35 cases of juvenile pemphigus vulgaris have been reported in the literature up until to now. During the pre‐steroid era, the outcome of the disease was usually regarded as fatal. Today, the prognosis has improved because of the possibility of early diagnosis by immunoflourescent ...
V, Graff-Lonnevig, T, Kaaman
openaire   +2 more sources

Neonatal pemphigus vulgaris

Journal of the American Academy of Dermatology, 1982
An infant was born with bullous lesions. The mother had had pemphigus vegetans diagnosed 1 year earlier, with remission during pregnancy. Histopathology and immunofluorescence of the infant's lesions were consistent with pemphigus vulgaris. The mother and infant had low titers of epidermal intercellular antibodies in their sera.
J S, Storer   +3 more
openaire   +2 more sources

Oesophageal Pemphigus vulgaris

Dermatology, 1988
Symptomatic involvement of the oesophageal mucosa by pemphigus vulgaris is rare. We describe 1 patient who was treated with oral steroids during a blistering phase, when epigastric pain developed. Endoscopy revealed multiple ulcerations all over the oesophagus, but gastroduodenal mucosa was normal.
H, Mobacken   +3 more
openaire   +2 more sources

Familial Pemphigus vulgaris

Dermatology, 1990
Familial pemphigus vulgaris was found in a Jewish woman and her son who developed the disease within a period of 18 months from one another. HLA typing was performed in the mother and son as well as in the unaffected father and sister. Examination of the HLA antigens in this family indicates that there may be different variants of DR4, DQw3 and that ...
V, Katzenelson   +5 more
openaire   +2 more sources

Familial Pemphigus vulgaris

Dermatology, 1985
A case of familial pemphigus vulgaris is described in an uncle and his niece who developed the disease 18 years apart. The man died from pemphigus in 1964. The diagnosis was confirmed histopathologically in both cases, but immunofluorescent microscopic studies were performed only in the woman because the technique was not available in 1964.
S, Brenner, B, Dorfman, M, Himelfarb
openaire   +2 more sources

LOCALIZED PEMPHIGUS VULGARIS*

The Journal of Dermatology, 1985
ABSTRACTA 32‐year‐old Japanese man was seen because of a crusted and oozing plaque on his scalp of 3 years' duration. Histologically, suprabasal clefts and bullae containing acantholytic cells as well as intercellular IgG and C3 deposits were found by the direct immunofluorescent technique. There were no circulating pemphigus antibodies. The lesion was
A, Yagi, S, Shiraishi, Y, Miki
openaire   +2 more sources

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