Results 141 to 150 of about 11,617 (181)
Variability of anti-PF4/heparin antibody results obtained by the rapid testing system ID-H/PF4-PaGIA [PDF]
Journal of Thrombosis and Haemostasis, 2009 Recent studies have shown that a low clinical pretest probability may be adequate for excluding heparin-induced thrombocytopenia. However, for patients with intermediate or high pretest probability, laboratory testing is essential for confirming or refuting the diagnosis.
Lorenzo Alberio +2 more
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Relation Of Heparin Binding And Conformation In PF4 AND LA-PF4 (βTG)
Thrombosis and Haemostasis, 1981Platelet factor 4 (PF4) and low affinity platelet factor 4 (LA-PF4) are platelet-specific secreted proteins that bind to heparin. β-thromboglobulin (βTG) appears to be derived from LA-PF4 by proteolysis of four NH2-terminal residues. PF4 and LA-PF4 (βTG) show 50% sequence homology including four cysteine residues and two pairs of lysine residues near ...
John C Holt +4 more
openaire +1 more source
Journal of the Chemical Society, Chemical Communications, 1974
It is shown by e.s.r. spectroscopy that a radical in γ-irradiated NH4PF6 and KPF6, previously identified as PF4, is in fact PF5–.
Shuddhodan P. Mishra +1 more
openaire +1 more source
It is shown by e.s.r. spectroscopy that a radical in γ-irradiated NH4PF6 and KPF6, previously identified as PF4, is in fact PF5–.
Shuddhodan P. Mishra +1 more
openaire +1 more source
Blood, 2009
Abstract Abstract 227 RUNX1/CBFA2 (Core binding factor A2) is a major transcription factor involved in hematopoiesis. RUNX1 mutations are associated with mild thrombocytopenia, platelet dysfunction, and predisposition to acute leukemia.
Kawalpreet Aneja +3 more
openaire +1 more source
Abstract Abstract 227 RUNX1/CBFA2 (Core binding factor A2) is a major transcription factor involved in hematopoiesis. RUNX1 mutations are associated with mild thrombocytopenia, platelet dysfunction, and predisposition to acute leukemia.
Kawalpreet Aneja +3 more
openaire +1 more source
Thrombosis and Haemostasis, 2015
SummaryShort chain polyphosphates (polyP) are pro-coagulant and pro-inflammatory platelet released inorganic polymers. The platelet chemokine platelet factor 4 (PF4) binds to lipid A on bacteria, inducing an antibody mediated host defense mechanism, which can be misdirected against PF4/heparin complexes leading to the adverse drug reaction heparin ...
S. Brandt +7 more
openaire +2 more sources
SummaryShort chain polyphosphates (polyP) are pro-coagulant and pro-inflammatory platelet released inorganic polymers. The platelet chemokine platelet factor 4 (PF4) binds to lipid A on bacteria, inducing an antibody mediated host defense mechanism, which can be misdirected against PF4/heparin complexes leading to the adverse drug reaction heparin ...
S. Brandt +7 more
openaire +2 more sources
Thrombosis Research, 1983
The recent introduction of the determinations of platelet factor 4 (PF4) and beta-thromboglobulin (beta TG) by radioimmunoassay provided a new tool to obtain knowledge of in vivo platelet activation. We evaluated the plasma level of PF4 and beta TG in 14 normal subjects (mean PF4 7.7 ng/ml; beta TG 28.8 ng/ml), in 29 patients with chronic stable ...
G, Cella +5 more
openaire +2 more sources
The recent introduction of the determinations of platelet factor 4 (PF4) and beta-thromboglobulin (beta TG) by radioimmunoassay provided a new tool to obtain knowledge of in vivo platelet activation. We evaluated the plasma level of PF4 and beta TG in 14 normal subjects (mean PF4 7.7 ng/ml; beta TG 28.8 ng/ml), in 29 patients with chronic stable ...
G, Cella +5 more
openaire +2 more sources
Journal of thrombosis and haemostasis : JTH
Heparin-induced thrombocytopenia (HIT), vaccine-induced immune thrombocytopenia and thrombosis (VITT), and VITT-like disorders are caused by anti-platelet factor 4 (PF4)/polyanion and/or anti-PF4 IgG antibodies. The rapid anti-PF4/polyanion and anti-PF4 chemiluminescence assays differentiate between typical HIT (anti-PF4/heparin) and VITT antibodies ...
Venkata A S, Dabbiru +11 more
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Heparin-induced thrombocytopenia (HIT), vaccine-induced immune thrombocytopenia and thrombosis (VITT), and VITT-like disorders are caused by anti-platelet factor 4 (PF4)/polyanion and/or anti-PF4 IgG antibodies. The rapid anti-PF4/polyanion and anti-PF4 chemiluminescence assays differentiate between typical HIT (anti-PF4/heparin) and VITT antibodies ...
Venkata A S, Dabbiru +11 more
openaire +1 more source
Chemokine PF4 Inhibits EV71 and CA16 Infections at the Entry Stage.
Journal of Virology, 2022Wenyan Zhang, Wenyan Zhang, Zhilei Zhao
exaly
Tetrafluorophosphite, PF4-, Anion
Journal of the American Chemical Society, 1994Karl O. Christe +5 more
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