Results 181 to 190 of about 15,097 (209)

BK Polyomavirus Infection in the Renal Transplant Recipient

Infectious Disease Clinics of North America, 2013
Renal transplant recipients continue to have progressive kidney dysfunction and renal graft loss has been attributed to emerging opportunistic infections, specifically BK virus (BKV). BKV is postulated to be selected by the new potent immunosuppressive medications and to be an important factor in graft failure.
Gayle P, Balba, Basit, Javaid, Joseph G, Timpone   +2 more
openaire   +2 more sources

Association of autism with polyomavirus infection in postmortem brains

Journal of Neurovirology, 2010
Autism is a highly heritable behavioral disorder. Yet, two decades of genetic investigation have unveiled extremely few cases that can be solely explained on the basis of de novo mutations or cytogenetic abnormalities. Vertical viral transmission represents a nongenetic mechanism of disease compatible with high parent-to-offspring transmission and with
Lintas, Carla, Altieri, Laura, Lombardi, Federica, Sacco, Roberto, Persico, Antonio M.   +4 more
openaire   +2 more sources

The role of sialic acid in human polyomavirus infections

Glycoconjugate Journal, 2006
JC virus (JCV) and BK virus (BKV) are human polyomaviruses that infect approximately 85% of the population worldwide [1,2]. JCV is the underlying cause of the fatal demyelinating disease, progressive multifocal leukoencephalopathy (PML), a condition resulting from JCV induced lytic destruction of myelin producing oligodendrocytes in the brain [3].
Gretchen V, Gee, Aisling S, Dugan, Natia, Tsomaia, Dale F, Mierke, Walter J, Atwood   +4 more
openaire   +2 more sources

[Urologic aspects of Polyomavirus infection].

Progres en urologie : journal de l'Association francaise d'urologie et de la Societe francaise d'urologie, 2010
JC virus (JCV) and BK virus (BKV) are human Polyomaviruses of the papovavirus family, which also includes a simian vacuolating virus 40 (SV40). Human Polyomaviruses were first isolated in 1971 from the brain (JCV) and urine (BKV) of two different patients. Human Polyomaviruses have a limited and specific tissue tropism infecting the renal tubular cells,
M, Thoulouzan, M, Courtade-Saidi, N, Kamar, L, Bellec, E, Huyghe, M, Soulié, P, Plante   +6 more
openaire   +1 more source

Post-transplantation polyomavirus infections.

Journal of nephrology, 1999
Increasing attention has been recently accorded to BK and JC viruses (BKV and JCV). Both these human polyomavirus (HPV) are members of the papovavirus family which includes the simian virus SV 40. BKV and JCV infect more than 60% of the population worldwide.
S, Boubenider, C, Hiesse, S, Marchand, A, Hafi, F, Kriaa, B, Charpentier   +5 more
openaire   +1 more source

Human Polyomavirus JC and BK Persistent Infection

2007
Primary contact with the human polyomaviruses (HPV) is followed by lifelong persistence of viral DNA in its host. The most prominent organs affected are the kidney, the Central Nervous System (CNS)and the hematopoietic system. Under impairment of immune competence limited activation of virus infection can be followed by prolonged virus multiplication ...
openaire   +2 more sources

Polyomavirus Infections

2020
Joseph Domachowske, Manika Suryadevara
openaire   +1 more source

The incidence of cytomegalovirus and BK polyomavirus infections in kidney transplant patients receiving mTOR inhibitors: A systematic review and meta‐analysis

Pharmacotherapy, 2023
Jiajia Yan, Changxi Wang, Kejing Tang
exaly  

Polyomavirus and cytomegalovirus infections are risk factors for grafts loss in simultaneous pancreas and kidney transplant

Transplant Infectious Disease, 2020
Fahad Aziz, Margaret R Jorgenson, Sandesh Parajuli   +2 more
exaly  

Infections à polyomavirus humains

EMC - Maladies infectieuses, 2009
H. Moret, V. Brodard
openaire   +1 more source