Results 51 to 60 of about 56,408 (287)

Infectious agents in atherosclerotic cardiovascular diseases through oxidative stress [PDF]

open access: yes, 2017
Accumulating evidence demonstrates that vascular oxidative stress is a critical feature of atherosclerotic process, potentially triggered by several infectious agents that are considered as risk co-factors for the atherosclerotic cardiovascular diseases (
Di Pietro, M   +4 more
core   +2 more sources

Placental Trophoblast Responses to Porphyromonas gingivalis Mediated by Toll-like Receptor-2 and -4

open access: yesJournal of Dentistry Indonesia, 2013
Trophoblast participates in preventing allorecognition and controlling pathogens that compromise fetal wellbeing. Toll-like receptors recognize conserved sequences on the pathogens surface and trigger effector cell functions.
Banun Kusumawardani   +3 more
doaj   +1 more source

Biofilm-stimulated epithelium modulates the inflammatory responses in co-cultured immune cells [PDF]

open access: yes, 2019
The gingival epithelium is a physical and immunological barrier to the microbiota of the oral cavity, which interact through soluble mediators with the immune cells that patrol the tissue at the gingival epithelium.
Bradshaw, David   +8 more
core   +4 more sources

Porphyromonas gingivalis in oral squamous cell carcinoma: A review.

open access: yesMicrobes and infection, 2021
Oral cancer contributes significantly to the global cancer burden. Oral bacteria play an important role in the spread of oral cancer, according to mounting evidence. The most proven instance is the carcinogenic implications of Porphyromonas gingivalis, a
Suchitra. Singh, A. K. Singh
semanticscholar   +1 more source

Melatonin Receptor Agonists as the "Perioceutics" Agents for Periodontal Disease through Modulation of Porphyromonas gingivalis Virulence and Inflammatory Response.

open access: yesPLoS ONE, 2016
Aim"Perioceutics" including antimicrobial therapy and host modulatory therapy has emerged as a vital adjunctive treatment of periodontal disease. Melatonin level was significantly reduced in patients with periodontal diseases suggesting melatonin could ...
Wei Zhou   +5 more
doaj   +1 more source

Optimization of quantitative polymerase chain reactions for detection and quantification of eight periodontal bacterial pathogens [PDF]

open access: yes, 2012
BACKGROUND: The aim of this study was to optimize quantitative (real-time) polymerase chain reaction (qPCR) assays for 8 major periodontal pathogens, i.e.
Cosyn, Jan   +8 more
core   +1 more source

Perkembangan Plasenta dan Pertumbuhan Janin pada Tikus Hamil yang Diinfeksi Porphyromonas Gingivalis Placental Development and Fetal Growth in Porphyromonas Gingivalis-Infected Pregnant Rats

open access: yesInsisiva Dental Journal, 2014
Maternal Porphyromonasgingivalis infection on periodontal tissue can result in Porphyromonasgingivalis dissemination to umbillical cord. Porphyromonas gingivalis presumably gain access to the systemic circulation via local tissue inflammation, and may ...
Banun Kusumawardani   +2 more
doaj   +1 more source

Preoperative serum immunoglobulin G and A antibodies to Porphyromonas gingivalis are potential serum biomarkers for the diagnosis and prognosis of esophageal squamous cell carcinoma

open access: yesBMC Cancer, 2018
Background The key-stone-pathogen, Porphyromonas gingivalis associates not only with periodontal diseases but with a variety of other chronic diseases such as cancer.
She-Gan Gao   +8 more
doaj   +1 more source

Porphyromonas Gingivalisin Different Peri-Implant Conditions: a Pilot Cross - Sectional Study

open access: yesActa Stomatologica Croatica, 2022
Objective: To assess the potential correlation between clinical peri-implant parameters and the presence of Porphyromonas gingivalis in different peri-implant conditions.
Nikolija Savčić   +4 more
doaj   +1 more source

Dysbiosis by neutralizing commensal mediated inhibition of pathobionts [PDF]

open access: yes, 2016
Dysbiosis in the periodontal microbiota is associated with the development of periodontal diseases. Little is known about the initiation of dysbiosis.
Bernaerts, Kristel   +6 more
core   +3 more sources

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