Results 171 to 180 of about 71,281 (314)

Treatment of periodontal disease for glycaemic control in people with diabetes [PDF]

, 2010
Al-Mubarak, Al-Zahrani, Aldridge, Almas, Altman, Armitage, Begg, Berlin, Braatvedt, Bunn, Calbacho, Campus, Chee, Christgau, Ciancio, Cohen, Copas, Cruz, Dag, Darré, Edwards, Egger, Faria-Almeida, Firlati, Florkowski, Goldstein, Grossi, Grossi, Halpern, Iwamoto, Janket, Jones, Katagiri, Khaw, Kiran, Lee, Lilford, Llambés, Llambés, Madden, Mansouri, Martorelli De Lima, Miedema, Miller, Milward, Navarro-Sanchez, O'Connell, Page, Papapanou, Promsudthi, Rocha, Rodrigues, Sacks, Safkan-Seppala, Sandberg, Sandberg, Santos, Sastrowijoto, Schara, Seppala, Seppälä, Smith, Sonoki, Spangler, Stewart, Stewart, Stratton, Taylor, Teeuw, Tervonen, Vergnes, Westfelt, Williams, Wolf, Yun   +74 more
core   +1 more source

Prions, Chaperones, and Proteostasis in Yeast.

Cold Spring Harbor Perspectives in Biology, 2017
T. Chernova, K. Wilkinson, Y. Chernoff
semanticscholar   +1 more source

In silico peptide self‐assembly reveals the importance of N‐terminal motifs and the inhibition mechanism of the mutation L38M in α‐synuclein fibrillation

Protein Science, Volume 35, Issue 4, April 2026.
Abstract Alpha‐synuclein (αSyn) is a presynaptic protein associated with several neurodegenerative diseases. While the non‐amyloid component (NAC) region of the αSyn sequence (residues 65–90) forms the core of all αSyn fibrils, recent findings suggest that the flanking regions play a key role in initiating or preventing amyloid formation. Two motifs in
Van T. T. Nguyen, Sabine M. Ulamec, David J. Brockwell, Sheena E. Radford, Carol K. Hall   +4 more
wiley   +1 more source

Prion Diseases [PDF]

Microbiology and Immunology, 1995
openaire   +3 more sources

NOTCH3 CADASIL Variant Receptor Aggregation Requires NOTCH3 Wild‐Type Receptors: Identification of Highly Selective Inhibitors That Block the Process

The FASEB Journal, Volume 40, Issue 6, 31 March 2026.
CADASIL is the major cause of early‐onset stroke and cognitive dysfunction, including dementia. It is caused by mutations in the NOTCH3 receptor that result in the formation of protein aggregates in the small vessel walls of the brain. We demonstrated that NOTCH3 CADASIL variant receptor aggregation is strictly dependent on interactions with NOTCH3 ...
Haijiang Wang, Xinxin Liu, Gido Gravesteijn, Alessia Atella, Jin Liu, Julie W. Rutten, Peter ten Dijke, Manuel A. F. V. Gonçalves, Saskia A. J. Lesnik Oberstein, David A. Baker   +9 more
wiley   +1 more source

PRION DISEASES [PDF]

Journal of Neurology, Neurosurgery & Psychiatry, 2004
R S G, Knight, R G, Will
openaire   +3 more sources

Doppel as an early‐stage biomarker promoting EMT and dissemination in ovarian cancers

International Journal of Cancer, Volume 158, Issue 6, Page 1755-1768, 15 March 2026.
What's new? Existing biomarkers for ovarian cancer (OC) lack the sensitivity and specificity needed to consistently distinguish the malignancy from non‐ovarian influences and other cancers. Here, the authors explored the ability of the prion‐like protein Doppel, normally found in the male testes, to detect OC.
Zulfikar Azam, Xiaojun Zhang, Riajul Wahab, Md Mahedi Hasan, Shivaniben Dhadhal, Bowon Kang, Md Mynul Hassan, Mazharul Karim, Jeong Uk Choi, Muhit Rana, Jian‐Ying Zhang, Sourav Roy, Youngro Byun, In‐San Kim, Jae Yun Song, Eugene P. Toy, Sireesha Y. Reddy, Farzana Alam, Taslim A. Al‐Hilal   +18 more
wiley   +1 more source

Prions, prions everywhere [PDF]

Nature Reviews Neuroscience, 2003
openaire   +1 more source

FAQs about mad cow disease and its impacts [PDF]

Animal ...
Jason Henderson
core  

Tau prions from Alzheimer’s disease and chronic traumatic encephalopathy patients propagate in cultured cells

Proceedings of the National Academy of Sciences of the United States of America, 2016
A. Woerman, A. Aoyagi, Smita Patel, S. A. Kazmi, I. Lobach, L. Grinberg, A. Mckee, W. Seeley, S. Olson, S. Prusiner   +9 more
semanticscholar   +1 more source