Results 301 to 310 of about 141,530 (342)
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Renin-Angiotensin-Aldosteron-System und Fibrinolyse
Medizinische Klinik, 2000Experimental, genetic and clinical evidence suggests that the renin-angiotensin-aldosterone system (RAAS) may participate in the pathogenesis of thromboembolic cardiovascular disorders such as coronary heart disease. This interrelationship may involve mechanisms other than changes in arterial blood pressure. In addition to various possible interactions,
K, Lottermoser +3 more
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Renin-angiotensin-aldosterone system
European Journal of Clinical Pharmacology, 1980There is increased activity of the renin, angiotensin, aldosterone (RAA) system in infancy and childhood. An inverse relationship between plasma renin, aldosterone and age has been demonstrated. In childhood hypertension due to renovascular disease or pyelonephritic scarring peripheral plasma renin is increased.
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The Renin-Angiotensin-Aldosterone System
S. Thatcher
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American Journal of Kidney Diseases, 2019
In experimental studies a low-protein diet (LPD) and renin-angiotensin-aldosterone system (RAAS) inhibitors are both reported to slow the progression of chronic kidney disease (CKD) and reduce proteinuria.
L. Koppe, D. Fouque
semanticscholar +1 more source
In experimental studies a low-protein diet (LPD) and renin-angiotensin-aldosterone system (RAAS) inhibitors are both reported to slow the progression of chronic kidney disease (CKD) and reduce proteinuria.
L. Koppe, D. Fouque
semanticscholar +1 more source
The Renin-Angiotensin-Aldosterone System
2019The renin—angiotensin—aldosterone system (RAAS) is one of the oldest hormonal systems—regardless of whether ‘old’ is defined as phylogenetically old, or in the sense of being discovered a long time ago. Originally, the RAAS has been described as a circulating endocrine system, with angiotensinogen being released by the liver, and renin secreted from ...
Steckelings, Ulrike Muscha +1 more
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[The renin-angiotensin-aldosterone system].
Deutsche medizinische Wochenschrift (1946), 1978"The present results indicate that there is a close relationship between the incretory function of the kidney, which is shown by the discharge of renin and similar materials, and the function of the adrenal cortex, especially the zona glomerulosa, which is the place of production of the sodium-retaining hormone aldosterone .
P, Vecsei, E, Hackenthal, D, Ganten
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Obesity and the renin– angiotensin–aldosterone system
Expert Review of Endocrinology & Metabolism, 2006The renin-angiotensin-aldosterone system (RAAS) is a key regulator of cardiovascular function. RAAS activity is upregulated in obesity despite concurrent renal sodium retention, which is a hallmark and principle determinant of obesity-associated hypertension.
Arya M, Sharma, Stefan, Engeli
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Renin-Angiotensin-Aldosterone System in Nephrotic Syndrome
American Journal of Kidney Diseases, 1984Previous studies on the renin-angiotensin-aldosterone system and fluid volumes in patients with nephrotic syndrome have not considered the nature of the underlying renal lesion. We compared plasma renin concentration (PRC), plasma aldosterone (PA), and plasma volume in three groups of patients: five nephrotic patients with minimal change disease on ...
T G, Hammond +5 more
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Adrenomedullin and the renin–angiotensin–aldosterone system
Regulatory Peptides, 2003Despite its positive inotropic effects and its propensity to stimulate the renin system, adrenomedullin (AM) is hypotensive as a result of dramatic reductions in peripheral resistance. Furthermore, it does not appear to increase aldosterone secretion in spite of often vigorous activation of circulating renin.
Christopher J, Charles +5 more
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The Renin-Angiotensin-Aldosterone System in Podocytes
Seminars in Nephrology, 2012The renin-angiotensin-aldosterone system (RAAS) plays a critical role in kidney function and its inhibition reduces proteinuria and preserves kidney function in patients with chronic kidney disease. Recent studies have shown that podocytes generate many components of the RAAS and they express receptors of RAAS, including angiotensin II ...
Dirk Oliver, Wennmann +2 more
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