Results 301 to 310 of about 141,530 (342)
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Renin-Angiotensin-Aldosteron-System und Fibrinolyse

Medizinische Klinik, 2000
Experimental, genetic and clinical evidence suggests that the renin-angiotensin-aldosterone system (RAAS) may participate in the pathogenesis of thromboembolic cardiovascular disorders such as coronary heart disease. This interrelationship may involve mechanisms other than changes in arterial blood pressure. In addition to various possible interactions,
K, Lottermoser   +3 more
openaire   +2 more sources

Renin-angiotensin-aldosterone system

European Journal of Clinical Pharmacology, 1980
There is increased activity of the renin, angiotensin, aldosterone (RAA) system in infancy and childhood. An inverse relationship between plasma renin, aldosterone and age has been demonstrated. In childhood hypertension due to renovascular disease or pyelonephritic scarring peripheral plasma renin is increased.
openaire   +2 more sources

The Role for Protein Restriction in Addition to Renin-Angiotensin-Aldosterone System Inhibitors in the Management of CKD.

American Journal of Kidney Diseases, 2019
In experimental studies a low-protein diet (LPD) and renin-angiotensin-aldosterone system (RAAS) inhibitors are both reported to slow the progression of chronic kidney disease (CKD) and reduce proteinuria.
L. Koppe, D. Fouque
semanticscholar   +1 more source

The Renin-Angiotensin-Aldosterone System

2019
The renin—angiotensin—aldosterone system (RAAS) is one of the oldest hormonal systems—regardless of whether ‘old’ is defined as phylogenetically old, or in the sense of being discovered a long time ago. Originally, the RAAS has been described as a circulating endocrine system, with angiotensinogen being released by the liver, and renin secreted from ...
Steckelings, Ulrike Muscha   +1 more
openaire   +3 more sources

[The renin-angiotensin-aldosterone system].

Deutsche medizinische Wochenschrift (1946), 1978
"The present results indicate that there is a close relationship between the incretory function of the kidney, which is shown by the discharge of renin and similar materials, and the function of the adrenal cortex, especially the zona glomerulosa, which is the place of production of the sodium-retaining hormone aldosterone .
P, Vecsei, E, Hackenthal, D, Ganten
openaire   +4 more sources

Obesity and the renin– angiotensin–aldosterone system

Expert Review of Endocrinology & Metabolism, 2006
The renin-angiotensin-aldosterone system (RAAS) is a key regulator of cardiovascular function. RAAS activity is upregulated in obesity despite concurrent renal sodium retention, which is a hallmark and principle determinant of obesity-associated hypertension.
Arya M, Sharma, Stefan, Engeli
openaire   +2 more sources

Renin-Angiotensin-Aldosterone System in Nephrotic Syndrome

American Journal of Kidney Diseases, 1984
Previous studies on the renin-angiotensin-aldosterone system and fluid volumes in patients with nephrotic syndrome have not considered the nature of the underlying renal lesion. We compared plasma renin concentration (PRC), plasma aldosterone (PA), and plasma volume in three groups of patients: five nephrotic patients with minimal change disease on ...
T G, Hammond   +5 more
openaire   +2 more sources

Adrenomedullin and the renin–angiotensin–aldosterone system

Regulatory Peptides, 2003
Despite its positive inotropic effects and its propensity to stimulate the renin system, adrenomedullin (AM) is hypotensive as a result of dramatic reductions in peripheral resistance. Furthermore, it does not appear to increase aldosterone secretion in spite of often vigorous activation of circulating renin.
Christopher J, Charles   +5 more
openaire   +2 more sources

The Renin-Angiotensin-Aldosterone System in Podocytes

Seminars in Nephrology, 2012
The renin-angiotensin-aldosterone system (RAAS) plays a critical role in kidney function and its inhibition reduces proteinuria and preserves kidney function in patients with chronic kidney disease. Recent studies have shown that podocytes generate many components of the RAAS and they express receptors of RAAS, including angiotensin II ...
Dirk Oliver, Wennmann   +2 more
openaire   +2 more sources

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