Results 221 to 230 of about 222,098 (349)
Regulation of PHOX2B gene expression by the long non‐coding natural antisense RNA PHOX2B‐AS1
PHOX2B is a transcription factor essential for autonomic nervous system development. We identify and characterize PHOX2B‐AS1, a human long non‐coding antisense transcript at the PHOX2B locus, along with its murine counterpart. Our findings reveal bidirectional transcription and reciprocal regulation: PHOX2B activates PHOX2B‐AS1, whereas PHOX2B‐AS1 ...
Simona Di Lascio +12 more
wiley +1 more source
Editorial Expression of Concern: Retinoic acid elicits cytostatic, cytotoxic and immunomodulatory effects on uveal melanoma cells. [PDF]
Vertuani S +5 more
europepmc +1 more source
Human RNase 7 is known to exert antimicrobial activity in epithelial tissues. Here, using SH‐SY5Y and U‐87 MG, neuroblastoma and glioblastoma cell lines, respectively, we found that RNase 7 enhances immune responses to LPS stimulation by reducing the expression of pro‐inflammatory cytokines, nitric oxide, and ROS.
Rosanna Culurciello +9 more
wiley +1 more source
Retinoic acid promotes expression of inflammatory factors in proliferative adult human heart cells. [PDF]
Gans IM +4 more
europepmc +1 more source
Estrogen Receptor β Activates the Human Retinoic Acid Receptorα -1 Promoter in Response to Tamoxifen and Other Estrogen Receptor Antagonists, but Not in Response to Estrogen [PDF]
Aihua Zou +6 more
openalex +1 more source
‘BJUI Clinical Dilemma’: Recurrent high‐grade non‐muscle‐invasive bladder cancer in 2026
BJU International, EarlyView.
Vignesh T. Packiam +10 more
wiley +1 more source
ABSTRACT Hematologic emergencies are urgent health conditions which result in significant mortality and morbidity unless timely therapeutic measures are taken. Therapeutic success depends on their timely and accurate recognition by hematology laboratory services.
Ganna Shestakova +2 more
wiley +1 more source
Effect of Maternal Administration with All-Trans Retinoic Acid on Lungs of Neonatal Pigs. [PDF]
Xiao X +9 more
europepmc +1 more source

