Results 71 to 80 of about 63,147 (208)
LINC02381 Promoted Cell Viability and Migration via Targeting miR-133b in Cervical Cancer Cells
Xiaohua Chen, Zhuxiang Zhang, Yan Ma, Hongxin Su, Peng Xie, Juntao Ran Department Of Radiation Therapy, First Hospital Of Lanzhou University, Lanzhou City, Gansu Province 730000, People’s Republic of ChinaCorrespondence: Juntao RanDepartment Of ...
Chen X +5 more
doaj
Convergence of Igf2 expression and adhesion signalling via RhoA and p38 MAPK enhances myogenic differentiation [PDF]
Cell-cell contact is essential for appropriate co-ordination of development and it initiates significant signalling events. During myogenesis, committed myoblasts migrate to sites of muscle formation, align and form adhesive contacts that instigate cell ...
Cobb, L.J. +8 more
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In chick embryo fibroblasts, the mRNA for extracellular matrix protein tenascin-C is induced 2-fold by cyclic strain (10%, 0.3 Hz, 6 h). This response is attenuated by inhibiting Rho-dependent kinase (ROCK).
Sarasa-Renedo, Ana +2 more
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Pengfei Li,1,* Huina Lv,2,* Min Xu,1 Bin Zang,1 Yegang Ma1 1Department of Thoracic Surgery, Cancer Hospital of China Medical University, Liaoning Cancer Hospital and Institute, Shenyang 110042, People’s Republic of China; 2Department of Ultrasound,
Li P, Lv H, Xu M, Zang B, Ma Y
doaj
RHOA GTPase Controls YAP-Mediated EREG Signaling in Small Intestinal Stem Cell Maintenance
Summary: RHOA, a founding member of the Rho GTPase family, is critical for actomyosin dynamics, polarity, and morphogenesis in response to developmental cues, mechanical stress, and inflammation.
Ming Liu +12 more
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Control of neurite outgrowth by RhoA inactivation [PDF]
J. Neurochem.(2012)120, 684–698.AbstractcAMP induces neurite outgrowth in the rat pheochromocytoma cell line 12 (PC12). In particular, di‐butyric cAMP (db‐cAMP) induces a greater number of primary processes with shorter length than the number induced by nerve growth factor (NGF).
Chan-Young, Jeon +11 more
openaire +2 more sources
Anillin Puts RhoA in Touch with PIP2 [PDF]
In this issue of Developmental Cell, Budnar and colleagues report how the scaffolding protein anillin uses cycles of transient binding interactions to enhance the residence time and signaling output of active RhoA to control actomyosin contractility at epithelial junctions and during cell division.
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From Mechanical Force to RhoA Activation [PDF]
Throughout their lives all cells constantly experience and respond to various mechanical forces. These frequently originate externally but can also arise internally as a result of the contractile actin cytoskeleton. Mechanical forces trigger multiple signaling pathways. Several converge and result in the activation of the GTPase RhoA. In this review we
Elizabeth C. Lessey +2 more
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RhoA and Rac1 as Mechanotransduction Mediators in Colorectal Cancer
Colorectal cancer (CRC) remains a leading cause of cancer-related deaths, creating an urgent need for innovative diagnostic solutions. Mechanobiology, a cutting-edge field that investigates how physical forces influence cell behavior, is now revealing ...
Nguyen, Nam-Trung +5 more
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Interaction between RhoA and ERK is mediated by the RhoA D-site.
(A) Co-immunoprecipitation of ERK and p-ERK with RhoA. COS-7 cells expressing GFP-RhoA were stimulated with EGF as indicated. GFP-RhoA was IPed from cell lysates with antibodies to GFP, and the co-IPed ERK and phosphor ERK (p-ERK) were analyzed by ...
Junfeng Tong (805234) +3 more
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