Results 41 to 50 of about 59,209 (214)
Peri-mortem arrhythmias in the non-cardiac intensive care unit. [PDF]
Nurs Crit CareAbstract Background Cardiovascular failure is recognized as a common final pathway at the end of life but there is a paucity of data describing terminal arrhythmias. Aim We aimed to describe arrhythmias recorded peri‐mortem in critically ill patients.
Okaj I +7 more
europepmc +2 more sources
Computing in Cardiology Conference (CinC), 2019 Atrial fibrillation (AF) is believed to shut down the normal function of sinoatrial node (SAN) by long-term overdrive suppression of its activity. Although AF-induced remodelling may impair SAN function, ionic mechanisms underlying sinus node dysfunction (SND) remain unclear.
Jieyun Bai +3 more
openalex +3 more sources
Japanese Circulation Journal, 1980 The diagnostic value of maximum corrected sinus node recovery time (CSNRT) and sinoatrial conduction time (SACT) was assessed in 15 patients with clinical evidence of sinus node (SN) dysfunction and 16 control subjects. The SACT was calculated by a modified method using only those sinus cycles with A1-A1 intervals falling within one standard deviation ...
Jin‐Jer Chen +3 more
openalex +5 more sources
Junctional Pacemaker May Replace the Sinoatrial Node
Cardiovascular Innovations and Applications, 2021 Junctional rhythm is usually seen in the clinic with different causes. We report a case of bicuspid aortic valve accompanied by sinoatrial node dysfunction.
Min Lin +4 more
doaj +1 more source
Cycle length restitution in sinoatrial node cells: a theory for understanding spontaneous action potential dynamics. [PDF]
PLoS ONE, 2014 Normal heart rhythm (sinus rhythm) is governed by the sinoatrial node, a specialized and highly heterogeneous collection of spontaneously active myocytes in the right atrium.
Patric Glynn, Birce Onal, Thomas J Hund
doaj +1 more source
Pathophysiology of Cav1.3 L-type calcium channels in the heart
Frontiers in Physiology, 2023 Ca2+ plays a crucial role in excitation-contraction coupling in cardiac myocytes. Dysfunctional Ca2+ regulation alters the force of contraction and causes cardiac arrhythmias. Ca2+ entry into cardiomyocytes is mediated mainly through L-type Ca2+ channels,
Sahil Zaveri +10 more
doaj +1 more source
Are physiological oscillations physiological?
The Journal of Physiology, EarlyView., 2023 Abstract figure legend Mechanisms and functions of physiological oscillations. Abstract Despite widespread and striking examples of physiological oscillations, their functional role is often unclear. Even glycolysis, the paradigm example of oscillatory biochemistry, has seen questions about its oscillatory function.
Lingyun (Ivy) Xiong, Alan Garfinkel
wiley +1 more source
Digital Technologies in Providing Development of Algorithms Surgical Treatment of Supraventricular Arrhythmias [PDF]
, 2006 The aim of the study was the development and clinical application of patient selection algorithm for surgical treatment of longlasting persistent atrial fibrillation. The study included 235 patients with acquired heart disease and coronary artery disease,
Melo +24 more
core +3 more sources
T-wave Inversion through Inhomogeneous Voltage Diffusion within the FK3V Cardiac Model [PDF]
Chaos 34, 043140 (2024), 2023 The heart beats due to the synchronized contraction of cardiomyocytes triggered by a periodic sequence of electrical signals called action potentials, which originate in the sinoatrial node and spread through the heart's electrical system. A large body of work is devoted to modeling the propagation of the action potential and to reproducing reliably ...
arxiv +1 more source