Results 221 to 230 of about 60,963 (256)
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Annual Review of Nutrition, 1988
INTR ODUCTI ON.... . . ......... . .. . . . . . . . ....... 483 HIST ORY OF THI AMIN . ..... . . . . . . . ... . .. .. . . . . . . . . . . . . . . . . . . . . . . .... . . . . . . . . . . . . . . .. ... . . . . . . . . . 484 BI OCHEMISTRY AN D MET AB OLISM 485 Biosynthesis . . . . . . . . . ..... . . . . . . . . . . . . . . . . . . . . . . . . .
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INTR ODUCTI ON.... . . ......... . .. . . . . . . . ....... 483 HIST ORY OF THI AMIN . ..... . . . . . . . ... . .. .. . . . . . . . . . . . . . . . . . . . . . . .... . . . . . . . . . . . . . . .. ... . . . . . . . . . 484 BI OCHEMISTRY AN D MET AB OLISM 485 Biosynthesis . . . . . . . . . ..... . . . . . . . . . . . . . . . . . . . . . . . . .
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Effect of Thiamin Deprivation on Thiamin Metabolism in Mice
The Journal of Nutrition, 1981Mice were made deficient by thiamin deprivation. Thiamin pyrophosphokinase and thiamin pyrophosphatase activities were measured, and their changes were compared to transketolase (TK), pyruvate and alpha-ketoglutarate dehydrogenase (DG) activities and thiamin pyrophosphate (TPP) level in the same tissues. Thiamin pyrophosphokinase activity was increased
Valentina G. Petushok+4 more
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Thiamine-quinone adducts. Colorimetric determination of thiamine
Experientia, 19772, 3-dichloro-1,4-naphtoquinone reacts with thiamine in the presence of NH3 to give a 1∶2 adduct, for which structureII is proposed on the basis of analytical and spectroscopic (IR) data. Such a reaction is very useful for thiamine quantitative determination.
V. De Mola, N. Gallo
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Simultaneous excretion of thiamine and thiamine phosphates by the dog
American Journal of Physiology-Legacy Content, 1962The fate of intravenously infused thiamine phosphates was studied in the dog. The degree of dephosphorylation by blood, tissue perfusion, and by the kidney was measured. Clearance determinations showed smaller values for these substances than those for creatinine.
Maurício Rocha E Silva+3 more
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2018
Starting with a brief history of beriberi and the discovery that thiamin deficiency is its cause, the symptoms and signs are reviewed. None are pathognomonic. The disease has a low mortality and a long morbidity. The appearance of the patient can be deceptive, often being mistaken for psychosomatic disease in the early stages.
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Starting with a brief history of beriberi and the discovery that thiamin deficiency is its cause, the symptoms and signs are reviewed. None are pathognomonic. The disease has a low mortality and a long morbidity. The appearance of the patient can be deceptive, often being mistaken for psychosomatic disease in the early stages.
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Prostaglandins, Leukotrienes and Essential Fatty Acids, 2005
After peroxynitrite addition to aqueous solutions of thiamine at neutral and alkaline pH formation of thiamine disulfide and fluorescent products was observed. The fluorescent compounds were identified as thiochrome (TChr) and oxodihydrothiochrome (ODTChr) using spectral and fluorescent methods as well as paper chromatography and mass spectrometry ...
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After peroxynitrite addition to aqueous solutions of thiamine at neutral and alkaline pH formation of thiamine disulfide and fluorescent products was observed. The fluorescent compounds were identified as thiochrome (TChr) and oxodihydrothiochrome (ODTChr) using spectral and fluorescent methods as well as paper chromatography and mass spectrometry ...
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Annals of Nutrition and Metabolism, 2012
Thiamin deficiency was long known as ‘beriberi’ in English and ‘kakké’ in Japan and China. The cause of beriberi was attributed to miasmas rising from wet soil and later to an unknown infectious organism. Systematic studies of beriberi began in the Dutch East Indies in the 1880s.
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Thiamin deficiency was long known as ‘beriberi’ in English and ‘kakké’ in Japan and China. The cause of beriberi was attributed to miasmas rising from wet soil and later to an unknown infectious organism. Systematic studies of beriberi began in the Dutch East Indies in the 1880s.
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Acta Crystallographica Section C Crystal Structure Communications, 1990
C 12 H 17 N 4 OS + •SCN − cristallise dans C2/c avec a=20,764, b=13,890, c=10,704 A, β=100,00°, Z=8; affinement jusqu'a R=0,044. C 12 H 17 N 4 OS + •SCN − •H 2 O cristallise dans P1 avec a=13,691, b=12,075, c=11,739 A, α=88,34, β=115,50, γ=106,61°, Z=4; affinement jusqu'a R=0,051.
A. Adeyemo+3 more
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C 12 H 17 N 4 OS + •SCN − cristallise dans C2/c avec a=20,764, b=13,890, c=10,704 A, β=100,00°, Z=8; affinement jusqu'a R=0,044. C 12 H 17 N 4 OS + •SCN − •H 2 O cristallise dans P1 avec a=13,691, b=12,075, c=11,739 A, α=88,34, β=115,50, γ=106,61°, Z=4; affinement jusqu'a R=0,051.
A. Adeyemo+3 more
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Thiamin-responsive Megaloblastic Anaemia: A Disorder of Thiamin Transport?
1984Thiamin-responsive megaloblastic anaemia (TRMA), associated with diabetes mellitus and sensorineural deafness (McKusick 24927) is a very rare disease, so far described in only five patients from four families. (Rogers et al., 1969; Viana and Carvalho, 1978; La Grutta et al., 1980; Haworth et al., 1982). We report the results of some studies carried out
V. Poggi+6 more
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Thiamine Concentration and Thiamine-destroying Activity in the Rumen of Cattle*)
Zentralblatt für Veterinärmedizin Reihe A, 2010Summary Data are presented on thiamine concentration and thiamine-destroying activity in rumen fluid. The total thiamine content ranged from about 300 to 600 nanograms per milliliter of rumen fluid, probably depending on the nature of the ingesta.
Etienne Muylle+3 more
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