Results 71 to 80 of about 233 (110)

Joint CB1 and NGF Receptor Activation Suppresses TRPM8 Activation in Etoposide-Resistant Retinoblastoma Cells. [PDF]

open access: yesInt J Mol Sci
Ludwiczak S   +7 more
europepmc   +1 more source
Some of the next articles are maybe not open access.

Related searches:

Differential inotropic-chronotropic action of thyronamine

European Journal of Pharmacology, 1973
Abstract J.R. BOISSIER, J.F. GIUDICELLI, S. LARNO and C. ADVENIER, Differential inotropic-chronotropic action of thyronamine , European J. Pharmacol. 22 (1973) 141–149. The cardiovascular and hemodynamic effects of thyronamine have been investigated in anesthetized dogs.
C Advenier, J R Boissier, J F Giudicelli
exaly   +3 more sources

Thyronamine, a new inotropic agent: its cardiovascular effects and mechanism of action.

Cardiovascular Research, 1974
Thyronamine, a decarboxylated, deiodinated derivative of thyroxine was administered to 21 anaesthetized dogs to assess its inotropic and chronotropic effects, to quantitate the haemodynamic response of a varying dosage schedule, and to investigate its mechanism of action on the cardiovascular system. All dogs received thyronamine at doses of 1, 2, 4, 8,
Donald C Harrison
exaly   +3 more sources

Cardiac effects of thyronamines

Heart Failure Reviews, 2008
3-Iodothyronamine (T(1)AM) is an endogenous compound derived from thyroid hormone through decarboxylation and deiodination, which interacts with a novel G protein-coupled receptor, known as trace amine-associated receptor 1 (TAAR1). TAAR1 and other receptors of this family are expressed in several tissues, including the heart.
Riccardo, Zucchi   +2 more
openaire   +2 more sources

Home - About - Disclaimer - Privacy