Results 201 to 210 of about 41,942 (251)

TGF-β1 relieves burn injury induced pain by alleviating inflammation in mouse. [PDF]

open access: yesPLoS One
Gao S   +8 more
europepmc   +1 more source

Bmscs loaded exosome hydrogel promotes the repair of rotator cuff injury in rats in vivo. [PDF]

open access: yesSci Rep
Peng K   +8 more
europepmc   +1 more source

Suppression of transforming growth factor-beta1 expression in keloids after cryosurgery

Cryobiology, 2017
The biological mechanism underlying cryosurgical treatment of keloids remains unclear. Transforming growth factor-beta1 (TGF-β1) has been implicated in the pathogenesis of keloids and was reported to be the target of several therapeutic modalities. However, the effect of cryosurgery on its expression in keloid tissue has not been yet investigated.
Sara M, Awad   +4 more
openaire   +4 more sources

Transforming Growth Factor β1: Implications in Adrenocortical Tumorigenesis

Endocrine Research, 2000
TGFbeta1, a multifunctional growth modulator, inhibits the proliferation of epithelial cells. TGFbeta1 signaling is dependent on the heterodimerization of the TGFbeta1 receptor II (TGFbeta1RII) with the TGFbeta1 receptor I (TGFbeta1RI). The cytoplasmic proteins Smads are the mediators of the TGFbeta1 signal.
ARNALDI G.   +4 more
openaire   +3 more sources

Expression of Transforming Growth Factor β1 in Ameloblastomas

Journal of Craniofacial Surgery, 2008
The aim of the present study was to compare the expression of transforming growth factor (TGF) beta1 in ameloblastomas (AMs) with different risk of recurrence by immunohistochemistry. A total of 29 cases of AMs were evaluated. The tumors were divided into 2 groups: group A (10 cases) composed of unicystic and peripheral AMs, associated with a low risk ...
IEZZI, GIOVANNA   +6 more
openaire   +3 more sources

Transforming growth factor-beta1 and regulators of apoptosis.

Annals of the New York Academy of Sciences, 2009
Growth inhibitory function of transforming growth factor-beta1 (TGF-beta1) is abolished in colorectal cancer cells as a consequence of mutations of various downstream signaling agents, such as p53, which fail to respond to TGF-beta1 stimulation. TGF-beta1 could also suppress T-cell-mediated anticancer immunity.
Stanislaw, Sulkowski   +3 more
openaire   +2 more sources

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