Combination of Markers, Including hsCRP, TGFβ 1, IL-6, and T-regs, and their Predictive Value as Early Warning Signs for Preeclampsia: Insights from a Tertiary Care Medical Centre in Eastern India. [PDF]
Dash P, Koppisetty BK, Jena SK, Nayak S.
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Growth factor and cell content in Platelet-Rich Plasma (PRP), Leukocyte- and Platelet-Rich Plasma (L-PRP), Platelet-Rich Fibrin (PRF) in patients with long bone defects from combat injuries. [PDF]
Husak V +5 more
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TGF-β1 relieves burn injury induced pain by alleviating inflammation in mouse. [PDF]
Gao S +8 more
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Bmscs loaded exosome hydrogel promotes the repair of rotator cuff injury in rats in vivo. [PDF]
Peng K +8 more
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Suppression of transforming growth factor-beta1 expression in keloids after cryosurgery
Cryobiology, 2017The biological mechanism underlying cryosurgical treatment of keloids remains unclear. Transforming growth factor-beta1 (TGF-β1) has been implicated in the pathogenesis of keloids and was reported to be the target of several therapeutic modalities. However, the effect of cryosurgery on its expression in keloid tissue has not been yet investigated.
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Transforming Growth Factor β1: Implications in Adrenocortical Tumorigenesis
Endocrine Research, 2000TGFbeta1, a multifunctional growth modulator, inhibits the proliferation of epithelial cells. TGFbeta1 signaling is dependent on the heterodimerization of the TGFbeta1 receptor II (TGFbeta1RII) with the TGFbeta1 receptor I (TGFbeta1RI). The cytoplasmic proteins Smads are the mediators of the TGFbeta1 signal.
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Expression of Transforming Growth Factor β1 in Ameloblastomas
Journal of Craniofacial Surgery, 2008The aim of the present study was to compare the expression of transforming growth factor (TGF) beta1 in ameloblastomas (AMs) with different risk of recurrence by immunohistochemistry. A total of 29 cases of AMs were evaluated. The tumors were divided into 2 groups: group A (10 cases) composed of unicystic and peripheral AMs, associated with a low risk ...
IEZZI, GIOVANNA +6 more
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Transforming growth factor-beta1 and regulators of apoptosis.
Annals of the New York Academy of Sciences, 2009Growth inhibitory function of transforming growth factor-beta1 (TGF-beta1) is abolished in colorectal cancer cells as a consequence of mutations of various downstream signaling agents, such as p53, which fail to respond to TGF-beta1 stimulation. TGF-beta1 could also suppress T-cell-mediated anticancer immunity.
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