Results 81 to 90 of about 7,937 (220)

Cardiac Amyloidosis: Mutimodality Imaging for Diagnosis and Prognosis

Journal of the Indian Academy of Echocardiography & Cardiovascular Imaging
Cardiac amyloid cardiomyopathy (CM) is a rapidly progressive disease that is frequently underrecognized and frequently diagnosed late in a significant number of individuals suffering from heart failure.
Nitin J. Burkule
doaj   +1 more source

T1 mapping in cardiac MRI [PDF]

, 2017
Quantitative myocardial and blood T1 have recently achieved clinical utility in numerous pathologies, as they provide non-invasive tissue characterization with the potential to replace invasive biopsy.
A Barison, A Liu, A Singh, AJ Taylor, AK McDiarmid, AS Flett, BB Mehta, BN Putko, C de Meester de Ravenstein, C Manisty, C Rapezzi, CWL Chin, CWL Chin, D Carrick, D Gensler, DC Look, Dina Radenkovic, DJ Pennell, DM Higgins, DM Higgins, DM Sado, DM Sado, DM Sado, DM Sado, DR Messroghli, E Dall'Armellina, F aus dem Siepen, F Ingravallo, F Weidemann, FL Ruberg, G Captur, G Captur, Gabriella Captur, GC Camargo, GS Slavin, H Xue, IS Syed, J Nadjiri, J Nadjiri, J Sanz, J Shao, J-P Carpenter, JA Luetkens, JAC Lima, James C. Moon, JC Moon, JC Wood, JCC Moon, JI Hamilton, JN Dungu, JPB O’Connor, K Chow, K Chow, K Hanneman, L Zhao, Lewis Ricketts, M Fontana, M Fontana, M Fontana, M Francone, M Lu, M Ugander, MG Friedrich, MH Alam, MS Sussman, MU Kamal, NC Edwards, P Haaf, P Kellman, P Kellman, P Kellman, P Kellman, P Kirk, P Lurz, PP Swoboda, R Deichmann, R Hinojar, R Hinojar, R Kaptein, R Kozor, RB Thompson, RJ Everett, S Badiani, S Bull, S Dass, S Lehrke, S Nordin, S Pica, S Roujol, S Roujol, S Weingärtner, S Weingärtner, S Weingärtner, Sebastian Weingärtner, SK Piechnik, SM Banypersad, SS Roujol, T Nishii, TA Treibel, TA Treibel, TD Karamitsos, U Blume, V Vassiliou, VH Urbieta-Caceres, VM Ferreira, VO Puntmann, YJ Hong, YY Chen   +107 more
core   +2 more sources

Reply: Bio-Profiling of Transthyretin Amyloid Cardiomyopathy.

JACC. Heart failure, 2021
Reply: Bio-Profiling of Transthyretin Amyloid ...
Rapezzi C., Sultan M. B., Maurer M. S.
openaire   +2 more sources

Genetic and Clinical Spectrum of Hereditary Transthyretin Amyloidosis in Brazil

Journal of the Peripheral Nervous System, Volume 31, Issue 1, March 2026.
ABSTRACT Background Transthyretin hereditary amyloidosis (ATTRv) clinical variability has been widely reported, not only across countries and variants but also among families and distinct regions within a single nation. One of the principal challenges in disease management is the accurate determination of age of onset (AOO), which is heterogeneous and ...
Gustavo Maximiano‐Alves, Carolina Lavigne‐Moreira, Marcus Vinicius Simões, Adilson Junior Pinto Galvão, Flavio Henrique Valicelli, Fernando Saraiva Coneglian, Elisa Vegezzi, Pedro Manoel Marques Garibaldi, Pedro José Tomaselli, Andrea Cortese, Wilson Marques Jr   +10 more
wiley   +1 more source

Cardiac Biomarker Change at 1 Year After Tafamidis Treatment and Clinical Outcomes in Patients With Transthyretin Amyloid Cardiomyopathy

Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease
Background Although tafamidis treatment improves prognosis in patients with wild‐type transthyretin amyloid cardiomyopathy, an optimal surrogate marker monitoring its therapeutic effect remains unclear.
Naoto Kuyama, Seiji Takashio, Tetsuya Oguni, Masahiro Yamamoto, Kyoko Hirakawa, Masanobu Ishii, Shinsuke Hanatani, Seitaro Oda, Yasushi Matsuzawa, Hiroki Usuku, Eiichiro Yamamoto, Toshinori Hirai, Mitsuharu Ueda, Kenichi Tsujita   +13 more
doaj   +1 more source

Contributions of Animal Models to the Mechanisms and Therapies of Transthyretin Amyloidosis

Frontiers in Physiology, 2019
Transthyretin amyloidosis (ATTR amyloidosis) is a fatal systemic disease caused by amyloid deposits of misfolded transthyretin, leading to familial amyloid polyneuropathy and/or cardiomyopathy, or a rare oculoleptomeningeal amyloidosis.
Ridwan Babatunde Ibrahim, Ridwan Babatunde Ibrahim, Yo-Tsen Liu, Yo-Tsen Liu, Yo-Tsen Liu, Yo-Tsen Liu, Ssu-Yu Yeh, Jin-Wu Tsai, Jin-Wu Tsai   +8 more
doaj   +1 more source

Familial amyloid polyneuropathy associated with TTRSer50Arg mutation in two Iberian families presenting a novel single base change in the mutant gene [PDF]

, 2007
Amyloid. 2007 Jun;14(2):147-52. Familial amyloid polyneuropathy associated with TTRSer50Arg mutation in two Iberian families presenting a novel single base change in the mutant gene.
COELHO, T, MASJUAN, J., MOREIRA, P., MUNAR‐QUÉS, M., SARAIVA, M.J., VIADER‐FARRÉ, C.   +5 more
core   +1 more source

Transthyretin cardiac amyloidosis: an update on diagnosis and treatment

ESC Heart Failure, 2019
Transthyretin cardiac amyloidosis (ATTR‐CA) demonstrates progressive, potentially fatal, and infiltrative cardiomyopathy caused by extracellular deposition of transthyretin‐derived insoluble amyloid fibrils in the myocardium.
Hiroyuki Yamamoto, Tomoki Yokochi
doaj   +1 more source

Initial experience with transcatheter tricuspid valve repair in patients with cardiac amyloidosis

ESC Heart Failure, 2023
Aims Wildtype transthyretin amyloid cardiomyopathy is an under‐recognized cause of heart failure in elderly patients. Transcatheter tricuspid valve repair is a newly emerging therapeutic option for severe tricuspid regurgitation (TR).
Isabel A. Hoerbrand, Martin J. Volz, Fabian Aus dem Siepen, Matthias Aurich, Philipp Schlegel, Nicolas A. Geis, Ute Hegenbart, Mathias H. Konstandin, Norbert Frey, Philip W. Raake   +9 more
doaj   +1 more source

Renal apolipoprotein A-I amyloidosis: a rare and usually ignored cause of hereditary tubulointerstitial nephritis [PDF]

, 2005
12openopenGregorini G; Izzi C; Obici L; Tardanico R; Röcken C; Viola BF; Capistrano M; Donadei S; Biasi L; Scalvini T; Merlini G; Scolari F.Gregorini, G; Izzi, C; Obici, L; Tardanico, R; Röcken, C; Viola, Bf; Capistrano, M; Donadei, S; Biasi, L; Scalvini,
BIASI, LUIGI, CAPISTRANO M., DONADEI S., GREGORINI G., IZZI C., MERLINI, GIAMPAOLO, OBICI L., ROCKEN C., SCALVINI T., SCOLARI F., TARDANICO R., VIOLA B. F.   +11 more
core   +1 more source