Background Vascular smooth muscle cell migration and accumulation in response to growth factors extensively contribute to the development of intimal thickening within the vessel wall.
van Breemen Cornelis +4 more
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Jak2 Tyrosine Kinase Mediates Oxidative Stress-induced Apoptosis in Vascular Smooth Muscle Cells [PDF]
Eric M. Sandberg, Peter P. Sayeski
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Defining the Role of Notch Signaling in Vascular Smooth Muscle Cell Differentiation [PDF]
accepted into the Wilbert C. Jordan Student Research Forum hosted by the Student National Medical Association at the 50th Annual Medical Education ConferenceComplications resulting from vascular disease such as atherosclerosis are a major health concern ...
Jackson, Kamari
core
Cysteinyl leukotriene signaling through perinuclear CysLT1 receptors on vascular smooth muscle cells transduces nuclear calcium signaling and alterations of gene expression [PDF]
Alison Eaton +4 more
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Functional preservation of vascular smooth muscle tissue [PDF]
The ionic and cellular feedback relationships operating to effect the vascular decompensatory modifications were examined to reveal procedures for implementing protective measures guarding against vascular collapse when returning from a weightless ...
Alexander, W. C. +2 more
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Imbalance of Endothelium-derived Relaxing and Contracting Factors: A New Concept in Hypertension? [PDF]
The endothelium has a strategical anatomical position between the circulating blood and vascular smooth muscle cells. It has recently been recognized that endothelial cells play an important regulatory role in the circulation.
Lüscher, T. F.
core
Heparin regulates the collagen phenotype of vascular smooth muscle cells: induced synthesis of an Mr 60,000 collagen. [PDF]
R A Majack, Paul Börnstein
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Intimal hyperplasia in blood vessels is primarily caused by the migration and proliferation of vascular smooth muscle cells. Excessive intimal thickening characterizes atherosclerosis as well as bypass graft and angioplasty failures.
Alison T. Stopeck M.D. +2 more
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Involvement of decreased myo‐inositol transport in lipopolysaccharide‐induced depression of phosphoinositide hydrolysis in vascular smooth muscle [PDF]
Yoko Sotoda +2 more
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Cbfa1 expression in vascular smooth muscle cells may be elevated by increased nitric oxide/iNOS
Maria Aparecida da Glória +4 more
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