Results 201 to 210 of about 75,185 (247)
Epstein‐Barr virus as a driver of lupus: Rethinking triggers in autoimmunity
Rheumatology &Autoimmunity, EarlyView.
Jiekai Tan, Fanlei Hu, Chuanhui Xu
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Herpes simplex virus latency and the immune response
Current Opinion in Microbiology, 1998Following infection, herpes simplex virus establishes latency in the nervous system and recurrences of lytic replication occur periodically. Molecular events which may determine how virus enters latency, how it is maintained and what occurs during reactivation have been investigated.
Barry T Rouse
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Clinics in Dermatology, 1984
The epidemiology of herpes simplex virus (HSV) infections in man has a number of unusual features (see Whitley, this volume). In particular, the recurrent lesions (herpes labialis, herpes genitalis, and herpes keratitis) often appear at the same peripheral site, the recurrences are often precipitated by particular stimuli, e. g., fever, excess exposure
T J, Hill +4 more
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The epidemiology of herpes simplex virus (HSV) infections in man has a number of unusual features (see Whitley, this volume). In particular, the recurrent lesions (herpes labialis, herpes genitalis, and herpes keratitis) often appear at the same peripheral site, the recurrences are often precipitated by particular stimuli, e. g., fever, excess exposure
T J, Hill +4 more
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Herpes simplex virus latency and nucleoside analogues
Antiviral Research, 1999Stacey Efstathiou +2 more
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Subclinical herpes virus reactivation and latency
Current Opinion in Infectious Diseases, 1998Although there has been considerable refinement in our understanding of the processes underlying the establishment and maintenance of latency, important research questions remain. Results from various workers imply that the establishment of latency may be a dynamic process and may offer possible therapeutic targets.
R, Patel, D R, Harper
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2001
MDV latency is defined as the persistence of the viral genome in the absence of production of infectious virus except during reactivation. A number of systems for studying MDV latency exist, and most involve the use of lymphoblastoid cells or tumors. It has been difficult to divorce latency and transformation.
R W, Morgan +6 more
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MDV latency is defined as the persistence of the viral genome in the absence of production of infectious virus except during reactivation. A number of systems for studying MDV latency exist, and most involve the use of lymphoblastoid cells or tumors. It has been difficult to divorce latency and transformation.
R W, Morgan +6 more
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A murine model of pseudorabies virus latency
Microbial Pathogenesis, 1992The mouse is a useful laboratory animal for studying various aspects of pseudorabies virus (PRV) virulence. Mice are highly susceptible hosts for PRV infection and are unable to survive acute viral infection. Because of this, mouse models have not been useful for studying PRV latent infections.
F A, Osorio, D L, Rock
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Expert Reviews in Molecular Medicine, 2003
Herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) are ubiquitous human pathogens. They share with other herpesviruses the ability to establish lifelong latent infection of the host. Periodic reactivation from latency is responsible for most of the clinical disease burden of HSV infection.
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Herpes simplex virus type 1 (HSV-1) and type 2 (HSV-2) are ubiquitous human pathogens. They share with other herpesviruses the ability to establish lifelong latent infection of the host. Periodic reactivation from latency is responsible for most of the clinical disease burden of HSV infection.
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Pseudorabies virus latency: restricted transcription
Archives of Virology, 1990Cloned pseudorabies virus (PRV) sequences representing over 80% of the viral genome were radiolabeled and individually hybridized to nucleic acid in the trigeminal ganglia of acutely and latently infected swine. In acutely infected animals, all cloned probes hybridized to PRV RNA and DNA.
J R, Lokensgard +2 more
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Latency and the tumour virus problem
Journal of Theoretical Biology, 1976Abstract Virus-cell relationships in latency and the association between oncogenic viruses and transformed cells have been compared in the light of Theobald Smith's dictum that “a damaging parasite tends to become a symbiont when the relationship between it, and its native host, has existed long”. This may be particularly significant in consideration
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